Results 231 to 240 of about 17,938 (260)

Non-canonical cell death in neurodegeneration: emerging mechanisms and therapeutic Frontiers. [PDF]

Apoptosis
Ercin N, Besli N, Beker M, Celik U.
europepmc   +1 more source

PANoptosis: a potential target of cardiomyopathy. [PDF]

Front Immunol
Sun M, Lu C, Wei J, Gao C, Dong D.
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Correction for Ma et al., Intercellular propagation of RIPK1/RIPK3 amyloid fibrils. [PDF]

Proc Natl Acad Sci U S A
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RIPK1 and RIPK3 in antibacterial defence

Biochemical Society Transactions, 2022
Upon sensing pathogenic bacterial infection, host cells activate a multitude of inflammatory and immunogenic responses to promote bacterial clearance and restore tissue homeostasis. RIPK1 and RIPK3 are two key players in antimicrobial defence, by either driving inflammatory signalling or inducing programmed cell death activation, ranging from apoptosis,
Hui Wen Yeap, Kaiwen W. Chen
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RIPK3 signaling and its role in the pathogenesis of cancers

Cellular and Molecular Life Sciences, 2021
RIPK3 (receptor-interacting protein kinase 3) is a serine/threonine-protein kinase. As a key component of necrosomes, RIPK3 is an essential mediator of inflammatory factors (such as TNFα-tumor necrosis factor α) and infection-induced necroptosis, a programmed necrosis.
Shanhui Liu, Mitchell F Denning, Jiwang Zhang   +2 more
exaly   +3 more sources

Regulatory mechanism of CaMKII δ mediated by RIPK3 on myocardial fibrosis and reversal effects of RIPK3 inhibitor GSK'872

Biomedicine and Pharmacotherapy, 2023
Myocardial fibrosis (MF) remains a prominent challenge in heart disease. The role of receptor-interacting protein kinase 3 (RIPK3)-mediated necroptosis is evident in the pathogenesis of numerous heart diseases. Concurrently, the activation of Ca2+/calmodulin-dependent protein kinase (CaMKII) is pivotal in cardiovascular disease (CVD).
Ji Cao, JiaNan Qian, Wei Zhang
exaly   +3 more sources

The role of RIPK3 in liver mitochondria bioenergetics and function

European Journal of Clinical Investigation, 2021
AbstractBackgroundReceptor‐interacting protein kinase 3 (RIPK3) is a key player of regulated necrosis or necroptosis, an inflammatory form of cell death possibly governing outcomes in chronic liver diseases, such as nonalcoholic fatty liver disease and nonalcoholic steatohepatitis.MethodsThis narrative review is based on literature search using PubMed ...
Tawhidul Islam, Marta B. Afonso, Cecília M. P. Rodrigues   +2 more
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Mechanisms of RIPK3‐induced inflammation

Immunology & Cell Biology, 2017
Receptor‐interacting protein kinase 3 (RIP3/RIPK3) is a multifunctional regulator of cell death and inflammation. It controls signalling downstream of the tumor necrosis factor (TNF) receptor family, DNA‐dependent activator of IFN‐regulatory factors (DAI) and toll‐like receptors (TLRs).
Inbar Shlomovitz, Sefi Zargrian, Motti Gerlic   +2 more
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