Results 181 to 190 of about 1,285,891 (313)
This study investigated how PYCR1 inhibition in bone marrow stromal cells (BMSCs) indirectly affects multiple myeloma (MM) cell metabolism and viability. Culturing MM cells in conditioned medium from PYCR1‐silenced BMSCs impaired oxidative phosphorylation and increased sensitivity to bortezomib.
Inge Oudaert +13 more
wiley +1 more source
Significance of Early Detection and Management of Trauma-Induced Inflammatory Root Resorption: A Case Report. [PDF]
Altahtam A +3 more
europepmc +1 more source
Bacteriophage as Related to the Root Nodule Bacteria of Alfalfa
S. C. Vandecaveye, H. Katznelson
openalex +1 more source
Using multi‐omic characterization, we aimed to identify key regulators specific to squamous cell lung carcinoma (SqCC). SqCC‐specific differentially expressed genes were integrated with metabolics data. High expression of the creatine transporter SLC6A8, along with elevated creatine levels, appeared to be a distinct metabolic feature of SqCC.
Johan Staaf +10 more
wiley +1 more source
Biocontrol efficacy of Pochonia chlamydosporia against root-knot nematode Meloidogyne javanica in eggplant and its impact on plant growth. [PDF]
Parveen A, Tanaka K, Khan M.
europepmc +1 more source
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser +14 more
wiley +1 more source
Starve or share? Phosphate availability shapes plant-microbe interactions. [PDF]
Schmidt M +4 more
europepmc +1 more source
Investigations on the root nodule bacteria of leguminous plants [PDF]
Artturi I. Virtanen, Tauno Laine
openalex +1 more source
Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu +18 more
wiley +1 more source

