Results 91 to 100 of about 68,287 (273)

Intraneural Hybrid Neurofibroma/ Schwannoma In Scalp: A Case Report [PDF]

open access: yesJournal of Clinical and Diagnostic Research, 2015
Benign Peripheral Nerve Sheath Tumours (BPNSTs) are traditionally classified into schwannoma, neurofibroma and perinurioma. Due to advances in molecular techniques, hybrid BPNSTs containing more than one histologic types have been documented.
Kishori Moni Panda, Naik Reena
doaj   +1 more source

The role of insulin-like growth factors signaling in merlin-deficient human schwannomas. [PDF]

open access: yes, 2012
Loss of the tumor suppressor merlin causes development of the tumors of the nervous system, such as schwannomas, meningiomas, and ependymomas occurring spontaneously or as part of a hereditary disease Neurofibromatosis Type 2 (NF2).
Ammoun, S   +7 more
core   +2 more sources

Identification of a distinct epigenetic subgroup with inferior PFS in intracranial mesenchymal tumors with FET::CREB fusion

open access: yesBrain Pathology, EarlyView.
Six new cases of intracranial mesenchymal tumors with FET::CREB fusion, alongside 20 previously reported cases underwent DNA methylation profiling uncovered a novel epigenetic subgroup distinguished by inferior PFS and unique clinicopathological, molecular, and epigenetic hallmarks.
Yong Lin   +12 more
wiley   +1 more source

Esophageal schwannoma [PDF]

open access: yesThe Journal of Thoracic and Cardiovascular Surgery, 2006
WOS ...
Basoglu, A   +3 more
openaire   +3 more sources

Meflin/ISLR is a meningeal cell‐specific marker involved in the development of meninges and meningioma progression

open access: yesBrain Pathology, EarlyView.
Mesenchymal stromal cell‐and fibroblast‐expressing Linx paralogue (Meflin) is expressed in embryonic meninges and contributes to meningeal homeostasis. In meningiomas, elevated Meflin correlates with higher grade and recurrence. Single‐cell RNA sequencing revealed a Meflin‐high tumor cell subset marked by reduced proliferation, WNT6 expression, and ...
Yukihiro Shiraki   +13 more
wiley   +1 more source

EVALUATION OF MONITORING OF PATIENTS WITH ACOUSTIC NEURINOMA

open access: yesArta Medica, 2020
Objectives. In 1974, in the United States of America, ”Florida Ear and Sinus Center” initiated a ”Wait & Scan” approach for acoustic neurinoma. It consists of monitoring vestibular schwannoma patients, during several years, using magnetic resonance ...
Marin Buracovschi   +5 more
doaj   +1 more source

The molecular pathogenesis of schwannomatosis, a paradigm for the co-involvement of multiple tumour suppressor genes in tumorigenesis [PDF]

open access: yes, 2016
Schwannomatosis is characterized by the predisposition to develop multiple schwannomas and, less commonly, meningiomas. Despite the clinical overlap with neurofibromatosis type 2 (NF2), schwannomatosis is not caused by germline NF2 gene mutations ...
Cooper, David Neil   +3 more
core   +1 more source

Liquid–Liquid Phase Separation in Major Hallmarks of Cancer

open access: yesCell Proliferation, EarlyView.
Aberrant condensates formed through phase separation are involved in the dysregulation of various critical cellular processes, including genome stability, transcriptional regulation and signal transduction, thereby promoting malignant transformation and the acquisition of multiple cancer hallmarks.
Chen‐chen Xie   +10 more
wiley   +1 more source

Schwannoma—A Rare Subepithelial Lesion of the Colon

open access: yesGE: Portuguese Journal of Gastroenterology, 2015
Schwannoma is a benign tumor arising from Schwann cells that form the neural sheath. Primary schwannoma of the colon is rare and a few cases have been reported.
Sandra Barbeiro   +9 more
doaj   +1 more source

Primary cerebellopontine angle melanocytoma: review. [PDF]

open access: yes, 2012
Introduction Primary cerebellopontine angle melanocytomas (PCPAMs) are very rare. Their natural history and prognosis are not fully understood. We reviewed the literature and add a new case to analyze PCPAM's presentation, radiological features, and ...
Elashaal, R   +3 more
core   +1 more source

Home - About - Disclaimer - Privacy