Results 121 to 130 of about 404 (147)
Ginkgetin from Ginkgo biloba: mechanistic insights into anticancer efficacy. [PDF]
Nat Prod BioprospectXiong B, Lu JJ, Guo H, Huang M, Li T.
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A Deep Dive into the Botanical and Medicinal Heritage of <i>Taxus</i>. [PDF]
Plants (Basel)Jîjie AR +10 more
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Ginkgolide B as a biopsychosocial treatment salvages repeated restraint stress-induced amygdalar anomalies in mice. [PDF]
IBRO Neurosci RepAdebayo OG +8 more
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Antidiabetic and Antigout Properties of the Ultrasound-Assisted Extraction of Total Biflavonoids from Selaginella doederleinii Revealed by In Vitro and In Silico Studies. [PDF]
Antioxidants (Basel)Gao Q +10 more
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Journal of Applied Toxicology, 2018
AbstractIncreased glycation of macromolecules via the reactive dicarbonyl and α‐oxoaldehyde methylglyoxal (MG) has shown an association with diabetes and its complications. In the present study, the protective effects of sciadopitysin against MG‐induced oxidative cell damage were investigated in the insulin‐producing pancreatic β‐cell line, RIN‐m5F ...
Kwang Sik Suh, Suk Chon, Eun Mi Choi
exaly +3 more sources
AbstractIncreased glycation of macromolecules via the reactive dicarbonyl and α‐oxoaldehyde methylglyoxal (MG) has shown an association with diabetes and its complications. In the present study, the protective effects of sciadopitysin against MG‐induced oxidative cell damage were investigated in the insulin‐producing pancreatic β‐cell line, RIN‐m5F ...
Kwang Sik Suh, Suk Chon, Eun Mi Choi
exaly +3 more sources
Sciadopitysin protects osteoblast function via its antioxidant activity in MC3T3-E1 cells
Food and Chemical Toxicology, 2013Age-related osteoblast dysfunction is the main cause of age-related bone loss in both men and women. In the present study, the effect of sciadopitysin, a type of biflavonoids, on osteoblast function was investigated in osteoblastic MC3T3-E1 cells.
Kwang Sik Suh, Eun Mi Choi
exaly +3 more sources
Journal of Biochemical and Molecular Toxicology, 2021
AbstractDiabetic cardiomyopathy (DCM), a diabetes complication, accounts for diabetes‐associated morbidity, mortality, and heart failure. Biflavonoids have been demonstrated to possess extensive pharmacological properties, such as antidiabetic and antioxidant activities. Our study aimed to explore the effects of sciadopitysin, a type of biflavonoid, on
Ben Liu
exaly +3 more sources
AbstractDiabetic cardiomyopathy (DCM), a diabetes complication, accounts for diabetes‐associated morbidity, mortality, and heart failure. Biflavonoids have been demonstrated to possess extensive pharmacological properties, such as antidiabetic and antioxidant activities. Our study aimed to explore the effects of sciadopitysin, a type of biflavonoid, on
Ben Liu
exaly +3 more sources
Free Radical Research, 2014
Methylglyoxal (MG) is a precursor of advanced glycation end products, which contribute to diabetic complications, including bone defects. In the present study, the effect of sciadopitysin on MG-induced cytotoxicity was investigated using osteoblastic MC3T3-E1 cells.
Sang Youl Rhee
exaly +3 more sources
Methylglyoxal (MG) is a precursor of advanced glycation end products, which contribute to diabetic complications, including bone defects. In the present study, the effect of sciadopitysin on MG-induced cytotoxicity was investigated using osteoblastic MC3T3-E1 cells.
Sang Youl Rhee
exaly +3 more sources
International Immunopharmacology, 2017
Previous studies reported that sciadopitysin (Sc), a type of biflavonoids, protects reactive oxygen species (ROS)-mediated osteoblast dysfunction, but its role in osteoclastogenesis remains unclear. In this study, we observed that Sc dose-dependently suppressed RANKL-induced osteoclastogenesis and bone resorption.
Qiang Lu, Ning Liu, Maolin Zhang
exaly +3 more sources
Previous studies reported that sciadopitysin (Sc), a type of biflavonoids, protects reactive oxygen species (ROS)-mediated osteoblast dysfunction, but its role in osteoclastogenesis remains unclear. In this study, we observed that Sc dose-dependently suppressed RANKL-induced osteoclastogenesis and bone resorption.
Qiang Lu, Ning Liu, Maolin Zhang
exaly +3 more sources
Journal of Applied Toxicology, 2021
AbstractThe accumulation of advanced glycation end products (AGEs) causes metabolic dysfunction and neuronal cell damage. Methylglyoxal (MG) is a major glycating agent that reacts with basic residues present in proteins and promotes the formation of AGEs.
Kwang Sik Suh +3 more
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AbstractThe accumulation of advanced glycation end products (AGEs) causes metabolic dysfunction and neuronal cell damage. Methylglyoxal (MG) is a major glycating agent that reacts with basic residues present in proteins and promotes the formation of AGEs.
Kwang Sik Suh +3 more
openaire +2 more sources