Results 301 to 310 of about 930,150 (335)

Nitric Oxide and Circulatory Shock

1998
Key discoveries in the past two decades have proved that the vascular endothelium is more than just a passive barrier between blood vessels and tissues, and have led to many important new concepts in vascular biology and pathophysiology.
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Circulatory mechanisms of shock and their mediators

Critical Care Medicine, 1987
Traditional concepts of shock therapy have been based on conventional monitoring. However, the availability of invasive monitoring systems has provided the means to describe the patterns of oxygen transport in various acute life-threatening illnesses.
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The role of lysosomes in circulatory shock

Life Sciences, 1976
Abstract Lysosomes are sensitive to the stressful stimuli which develop in the shock states (i.e., ischemia, hypoxia, acidosis). As a result, lysosomal membranes become leaky and tissue lysosomes swell. These conditions are favorable for the leakage of lysosomal contents, largely acid hydrolases, into the cytoplasm of splanchnic cells, particularly ...
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Circulatory shock in pregnant sheep

American Journal of Obstetrics and Gynecology, 1974
Uteroplacental and fetal hemodynamics and oxygen transfer were studied in near-term pregnant sheep during progessively induced hemorrhagic shock and blood reinfusion. When the perfusing pressure fell to 50 or 60 mm. Hg, uteroplacental vascular resistance increased significantly and the blood flow fell more than the arterial pressure.
Charles R. Brinkman, Massoud Mofid, Nicholas S. Assali   +2 more
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Anesthetic Techniques in Circulatory Shock [PDF]

, 1987
This review will focus on the cardiovascular effects of the most common anesthetics, with special reference to the anesthesia of the shocked patient.
P. Van der Linden, E. Gilbart
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Microaxial Flow Pump or Standard Care in Infarct-Related Cardiogenic Shock.

New England Journal of Medicine
BACKGROUND The effects of temporary mechanical circulatory support with a microaxial flow pump on mortality among patients with ST-segment elevation myocardial infarction (STEMI) complicated by cardiogenic shock remains unclear.
J. Møller, Thomas Engstrøm, L. O. Jensen, H. Eiskjær, N. Mangner, Amin Polzin, P. C. Schulze, C. Skurk, P. Nordbeck, P. Clemmensen, V. Panoulas, Sebastian Zimmer, Andreas Schäfer, Nikos Werner, M. Frydland, L. Holmvang, J. Kjærgaard, Rikke Sørensen, J. Lønborg, M. Lindholm, N. J. Udesen, Anders Junker, H. Schmidt, C. Terkelsen, Steffen Christensen, E. H. Christiansen, Axel Linke, F. Woitek, Ralf Westenfeld, Sven Möbius-Winkler, Kristian Wachtell, H. Ravn, J. Lassen, Søren Boesgaard, O. Gerke, Christian Hassager   +35 more
semanticscholar   +1 more source

Critical care ultrasonography in circulatory shock

Current Opinion in Critical Care, 2017
Purpose of review The objective was to define the role of ultrasound in the diagnosis and the management of circulatory shock by critical appraisal of the literature. Recent findings Assessment of any patient's hemodynamic profile based on clinical examination can be sufficient in ...
Geert Koster, Iwan C. C. van der Horst
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Prostaglandin metabolism during circulatory shock

Biochimica et Biophysica Acta (BBA) - General Subjects, 1977
Abstract The rates of metabolic degradation and the patterns of metabolite formation of tritium-labeled prostaglandins E2 and F2α were assessed in vitro in tissues obtained from normal rabbits and from rabbits subjected to hemorrhagic or endotoxic shock. Normal rabbit tissues metabolized prostaglandin E2 at the following rates: renal cortex 479 ± 34,
John T. Flynn, Allan M. Lefer
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Microvascular Fluid Resuscitation in Circulatory Shock

Nursing Clinics of North America, 2017
The microcirculation is responsible for blood flow regulation and red blood cell distribution throughout individual organs. Patients with circulatory shock have acute failure of the cardiovascular system in which there is insufficient delivery of oxygen to meet metabolic tissue requirements.
Shannan K. Hamlin, La Donna Y. Christy, Hsin Mei Chen, Penelope Z. Strauss   +3 more
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The Circulatory Defect of Septic Shock

1987
Septic shock in man is generally characterized by a normal or mostly increased cardiac output (CO), a decreased systemic vascular resistance (SVR), a decreased arterio-mixed venous oxygen content difference \((Ca - \bar{v}{{O}_{2}})\) and elevated blood lactate levels [1–9].
A. B. J. Groeneveld, L. G. Thijs
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