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2011
Shock is a common manifestation of many forms of critical illness. Although a patient with hypotension can have shock, shock is not necessarily defined by hypotension. That is, a patient can have a “normal” blood pressure and have shock concurrently.
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Shock is a common manifestation of many forms of critical illness. Although a patient with hypotension can have shock, shock is not necessarily defined by hypotension. That is, a patient can have a “normal” blood pressure and have shock concurrently.
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Mechanical Ventilatory Support in Circulatory Shock
Critical Care Clinics, 1993Mechanical support of ventilation is associated with a number of hemodynamic consequences that result from direct and indirect hydrostatic phenomena, humoral effects, and changes in distribution of peripheral blood flow, particularly in patients with circulatory shock.
Dennis M. Greenbaum, Guy E. DeGent
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Circulatory shock associated with purulent peritonitis
The American Journal of Surgery, 1981The relative roles of bacterial infection, fluid loss and myocardial failure were investigated in 24 patients in whom circulatory shock appeared as a complication of purulent peritonitis. The 13 acute survivors, including 6 hospital survivors, had strikingly lower initial plasma volumes and total blood volumes than the 11 patients who died. Differences
Vincent, Jean Louis +3 more
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Endorphins in circulatory shock
Critical Care Medicine, 1988Bart Chernow, Lena M. Napolitano
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A rare cause of circulatory shock
Anadolu Kardiyoloji Dergisi/The Anatolian Journal of Cardiology, 2014Cihan Altin +4 more
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Circulatory Shock: A Microcirculatory Defect
1985The pathophysiological defect which characterizes shock is that of perfusion failure in which blood flow to maintain the function of vital organ systems is critically reduced. As a clinical syndrome, it is characterized by protracted prostration, pallor, coldness and moistness of the skin, collapse of the superficial veins, alterations in pressure is ...
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Altered Mitochondrial Metabolism in Circulatory Shock
1977Studies of mitochondrial function after circulatory shock, hemorrhagic or endotoxic, have revealed defective oxidative metabolism in several organs, such as the liver, kidney, and the brain (1,2,3). In this comment I would like to emphasize certain important aspects of these findings.
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Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature, 2020
N. Kapur
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N. Kapur
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