Results 151 to 160 of about 13,228 (260)

Effect of chronic high‐altitude hypoxia on retinal vascular phenotype, choroidal and retinal vasoreactivity in Peruvian highlanders

open access: yesExperimental Physiology, EarlyView.
Abstract High‐altitude chronic hypoxia can induce excessive erythrocytosis (EE, defined as a haemoglobin concentration of ≥21 g/dL in men), leading to hyperviscosity and promoting endothelial dysfunction. We aimed to assess whether EE affects the retinal vascular phenotype and the ophthalmological vascular response to CO2.
Aymeric Paillisser   +12 more
wiley   +1 more source

Returning to Performance After ACL Injury in Competitive Alpine Skiing: A Scoping Review and Evidence- and Expert-Informed Practice Recommendations. [PDF]

open access: yesScand J Med Sci Sports
Spörri J   +10 more
europepmc   +1 more source

Altitude or heat training to increase haemoglobin mass and endurance exercise performance in elite sport

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Upper panel, high‐altitude training typically encompasses 3–4 weeks of altitude exposure combined with training either at altitude or at sea level. Following this, a response for haemoglobin mass (Hbmass) is seen in two of three studies, which coincides with some performance gains in <50% of studies.
Carsten Lundby, Paul Robach
wiley   +1 more source

Intramuscular pathways of maladaptation in overtraining syndrome

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend The transition from adaptive overreaching to maladaptive overtraining and mechanisms through which excessive training load can lead to performance decline. Four interconnected pathophysiological domains are highlighted: neural fatigue, involving both central and peripheral components such as altered sensory feedback and reflex ...
Emily Shorter   +4 more
wiley   +1 more source

Mechanisms of haemoglobin mass expansion following heat stress

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Theorised mechanisms of heat stress‐induced erythropoiesis which may facilitate the expansion of haemoglobin mass. A, acute: heat stress‐induced renal ischaemia. Redistribution of blood flow away from the kidneys to facilitate thermoregulation during heat stress could reduce renal oxygen delivery, subsequently lowering renal PO2${
Elliott J. Jenkins   +4 more
wiley   +1 more source

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