Results 251 to 260 of about 1,535,898 (372)

Myogenin regulates denervation‐dependent muscle atrophy in mouse soleus muscle

open access: yesJournal of Cellular Biochemistry, 2011
P. Macpherson, Xun Wang, D. Goldman
semanticscholar   +1 more source

Mechanical factors in the initiation of eccentric contraction‐induced injury in rat soleus muscle.

open access: green, 1993
Gordon L. Warren   +3 more
openalex   +1 more source

APE1/Ref‐1 inhibition via APX3330 lowers monocyte/macrophage infiltration without ameliorating the structure and function of dystrophic mdx hindlimb muscles

open access: yesPhysiological Reports, Volume 13, Issue 15, August 2025.
Abstract Chronic inflammation and oxidative stress exacerbate muscle wasting and weakness in Duchenne muscular dystrophy (DMD). Apurinic/apyrimidinic endonuclease 1/redox factor‐1 (APE1/Ref‐1) regulates transcription factors involved in inflammatory and oxidative stress pathways.
Hannah Lalunio   +7 more
wiley   +1 more source

Effects on venous flow of transcutaneous electrical stimulation, neuromuscular stimulation, and sham stimulation on soleus muscle: A randomized crossover study in healthy subjects. [PDF]

open access: yesMedicine (Baltimore), 2022
Senin-Camargo F   +5 more
europepmc   +1 more source

Materials fatigue initiates eccentric contraction‐induced injury in rat soleus muscle.

open access: green, 1993
Gordon L. Warren   +4 more
openalex   +2 more sources

Aldehyde Oxidase 1 Deficiency Enhances Aerobic Exercise Performance by Promoting Skeletal Muscle Adaptation and Improving Mitochondrial Function

open access: yesThe FASEB Journal, Volume 39, Issue 14, 31 July 2025.
Aox1 expression is negatively associated with the oxidative muscle fiber phenotype and is downregulated by endurance training. Aox1 deficiency enhances aerobic exercise capacity in mice, at least in part, through the upregulation of PGC‐1α, which promotes oxidative fiber‐type transition, boosts mitochondrial oxidative phosphorylation, and increases ...
Yan Liu   +10 more
wiley   +1 more source

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