Results 131 to 140 of about 19,768,754 (339)
Chapter 4: Always “Now” is the Peak Moment
Biophysics and Physicobiology, 2023 Editorial team for the Special Issue on Oosawa’s Lectures
doaj +1 more source
Molecular Oncology, EarlyView.This study integrates publicly available transcriptomic datasets to identify molecular signatures associated with response to neoadjuvant chemoradiotherapy in locally advanced rectal cancer. By analyzing a combination of multiple cohorts with bioinformatics approaches, we reveal biological pathways and immune‐related features that may improve ...
Aleksandra Stanojevic +10 more
wiley +1 more source
Editorial: English translation of “The Oosawa Lectures on DIY Statistical Mechanics”
Biophysics and Physicobiology, 2021 Editorial team for the Special Issue on Oosawa’s Lectures
doaj +1 more source
The VHL tumor suppressor at the crossroad of protein folding, aggregation, and cancer
Molecular Oncology, EarlyView.Mutations, environmental stress, and chaperone dysfunction can destabilize pVHL, promoting its conversion from the native folded state into amyloid‐like assemblies. This transition may contribute to protein storage, cell dormancy, survival, and drug resistance.
Lara Abad +2 more
wiley +1 more source
Part II. Application of Statistical Mechanics in Biological Phenomena6.1
Biophysics and Physicobiology, 2023 Editorial team for the Special Issue on Oosawa’s Lectures
doaj +1 more source
Molecular Oncology, EarlyView.This study identifies ARHGAP5, in addition to the frequently mutated ARHGAP35, as significantly mutated in endometrial cancer. Mutations in both genes co‐occur and are associated with their correlated downregulation. Functional CRISPR studies show that both paralogs regulate similar pathways, including actin cytoskeleton organization.
Mathilde Pinault +12 more
wiley +1 more source
Feminist Encounters: Special Issue on Slow Intimacy [PDF]
Editorial: Special Issue on Slow ...
Lou-Marié Kruger, Amanda Gouws
core +2 more sources
Molecular Oncology, EarlyView.The kinase SRPK1 directly interacts with the protein TOPBP1 and regulates the pre‐mRNA splicing of WIZ thereby contributing to the activation of the ATR/CHK1 replicative checkpoint in response to replicative stress. This allows cancer cells' genomic stability and survival.
Amani Shreim +17 more
wiley +1 more source