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Imaging in spondyloarthropathies
Current Rheumatology Reports, 2004Seronegative spondyloarthropathies comprise a group of inflammatory arthritides, which consists of ankylosing spondylitis, psoriatic arthritis, reactive arthritis (Reiter's syndrome), enteropathic arthritis, and undifferentiated spondyloarthropathy. All of them share common laboratory, clinical, and imaging findings, with characteristic involvement of ...
Mikayel, Grigoryan +3 more
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Genetics of the spondyloarthropathies
Current Opinion in Rheumatology, 1991Following three decades of study of the genetics of the spondyloarthropathies by segregation analysis of families, research has increasingly made use of new methods of molecular genetics and immunology. In the past year, most attention has been given to the structure and function of HLA-B27, which, with its various subtypes, is the major phenotype ...
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Enthesitis in spondyloarthropathy
Current Opinion in Rheumatology, 1999Inflammation at the insertions of ligaments, tendons, or joint capsules to bone, which is termed enthesitis, is a characteristic feature of spondyloarthropathy. Because of the relative inaccessibility of the enthesis, the inflammatory, microbiologic, and immunologic events at that site have been poorly defined. Recent magnetic resonance imaging studies
D, McGonagle +5 more
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Classification of the spondyloarthropathies
Current Opinion in Rheumatology, 2005The spondyloarthropathies are a group of conditions which share similar clinical features. Classification criteria permit separation of the conditions, allow better targeting of therapies, better measurement of outcomes, and better prognostic information.
Paul J, Healy, Philip S, Helliwell
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The Seronegative Spondyloarthropathies
Primary Care: Clinics in Office Practice, 2018The seronegative spondyloarthropathies are a group of five diseases characterized by inflammatory oligoarticular arthritis, enthesitis, and axial involvement. They have an increased incidence of the HLA-B27 gene. They are commonly associated with extra-articular features including involvement of the skin, eyes, and gastrointestinal tract.
Ayyappa S, Duba, Stephanie D, Mathew
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Spondyloarthropathy in the Jurassic
The Lancet, 20021454 THE LANCET • Vol 360 • November 9, 2002 • www.thelancet.com 14 Tchernina N. Rising unemployment and coping strategies: the case of the Novosibirsk oblast in Russia. In: Cornia GA, Paniccia R, eds. The mortality crisis in transitional economies. New York: Oxford University Press, 2000: 151–73. 15 Nemtsov A. Estimates of total alcohol consumption in
Bruce, Rothschild +2 more
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IMMUNOGENETICS OF SPONDYLOARTHROPATHIES
Rheumatic Disease Clinics of North America, 1992The association of HLA-B27 with ankylosing spondylitis and related spondyloarthropathies has been known for two decades and has provided a great impetus to the epidemiologic studies and also helped broaden the clinical spectrum of these diseases.
M A, Khan, H, Kellner
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UNDIFFERENTIATED SPONDYLOARTHROPATHIES
Rheumatic Disease Clinics of North America, 1992The term undifferentiated spondyloarthropathy (uSpA) refers to patients with clinical and roentgenographic features suggestive of spondyloarthropathies but not fulfilling the diagnostic or classification criteria for any of the currently established disease categories.
H, Zeidler, W, Mau, M A, Khan
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Seronegative Spondyloarthropathies
Therapeutische Umschau, 2002Die Gruppe der seronegativen Spondyloarthropathien ist gekennzeichnet durch eine unilaterale oder bilaterale Sakroiliitis und eine Spondylitis, mit oder ohne periphere Polyarthritis. Vergesellschaftet sind die skelettalen Befunde mit extraartikulären Manifestationen verschiedener Art. Es besteht eine Assoziation zum HLA-B27-Antigen.
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The genetics of spondyloarthropathies
Joint Bone Spine, 2006The spondyloarthropathies constitute a group of inflammatory joint diseases linked by shared characteristics that include a strong common genetic background. Genetic factors include major histocompatibility complex (MHC) genes, among which HLA-B27 contributes 30% of the overall genetic susceptibility to spondyloarthropathies, and non-MHC genes, none of
Maxime, Breban +4 more
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