Mechanisms for the formation of active sites in single-atom alloys.
Karageorgiou I, Michaelides A, Berger F.
europepmc +1 more source
Transcriptome assemblies for two drug-type cannabis chemotypes by long-read RNA sequencing. [PDF]
Berkowitz O +5 more
europepmc +1 more source
Tumors contain diverse cellular states whose behavior is shaped by context‐dependent gene coordination. By comparing gene–gene relationships across biological contexts, we identify adaptive transcriptional modules that reorganize into distinct vulnerability axes.
Brian Nelson +9 more
wiley +1 more source
Measurement Properties and Minimal Detectable Change of the 6-Min Step Test for Estimating Cardiorespiratory Fitness in Hospitalized Individuals After Stroke. [PDF]
Aguiar Pavesi RO +3 more
europepmc +1 more source
RIPK4 function interferes with melanoma cell adhesion and metastasis
RIPK4 promotes melanoma growth and spread. RIPK4 levels increase as skin lesions progress to melanoma. CRISPR/Cas9‐mediated deletion of RIPK4 causes melanoma cells to form less compact spheroids, reduces their migratory and invasive abilities and limits tumour growth and dissemination in mouse models.
Norbert Wronski +9 more
wiley +1 more source
Supporting Change in Autism Services: Bridging the Gap between Theory and Practice
Reem Mouawad
doaj +1 more source
Assessing Statistical Methods for Estimating the Cerebrospinal Fluid Early Fungicidal Activity as an Endpoint for Cryptococcal Meningitis Clinical Trials: A Systematic Review and Simulation Study. [PDF]
Fuszard M, Boulware DR, Dai B.
europepmc +1 more source
COMP–PMEPA1 axis promotes epithelial‐to‐mesenchymal transition in breast cancer cells
This study reveals that cartilage oligomeric matrix protein (COMP) promotes epithelial‐to‐mesenchymal transition (EMT) in breast cancer. We identify PMEPA1 (protein TMEPAI) as a novel COMP‐binding partner that mediates EMT via binding to the TSP domains of COMP, establishing the COMP–PMEPA1 axis as a key EMT driver in breast cancer.
Konstantinos S. Papadakos +6 more
wiley +1 more source

