Results 131 to 140 of about 120,735 (166)
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Otolaryngologic Clinics of North America, 1989
The author reviews the etiology of subglottic stenosis. The diagnosis, including the use of radiology and endoscopy, is covered. Treatment of both congenital and acquired forms of subglottic stenosis is discussed.
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The author reviews the etiology of subglottic stenosis. The diagnosis, including the use of radiology and endoscopy, is covered. Treatment of both congenital and acquired forms of subglottic stenosis is discussed.
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Otolaryngologic Clinics of North America, 1984
Severe subglottic stenosis or stenosis that persists after a number of repairs have failed can be treated by an operation to widen the entire cricoid. This is accomplished by splitting the cricoid arch anteriorly and the posterior cricoid lamina posteriorly, both in the vertical midline.
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Severe subglottic stenosis or stenosis that persists after a number of repairs have failed can be treated by an operation to widen the entire cricoid. This is accomplished by splitting the cricoid arch anteriorly and the posterior cricoid lamina posteriorly, both in the vertical midline.
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MANAGEMENT OF SUBGLOTTIC STENOSIS
Otolaryngologic Clinics of North America, 2000Evaluation and management of pediatric subglottic stenosis is complex and technically challenging. Laryngotracheal reconstruction has become the standard of care for symptomatic subglottic stenosis in children. Careful preoperative planning, meticulous surgery, and dedicated postoperative care are essential for a successful outcome.
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Histopathology of congenital subglottic stenosis
The Laryngoscope, 1979AbstractThree patients with congenital subglottic stenosis are presented and whole organ serial‐section studies of their larynges are discussed. A superiorly displaced first tracheal ring is observed to form a cartilaginous subglottic stenosis in one. This “trapped first ring” is demonstrated in horizontal, sagittal and coronal planes.
G F, Tucker +3 more
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Acquired Subglottic Stenosis in Children
Annals of Otology, Rhinology & Laryngology, 1987Subglottic stenosis is a disorder characterized by narrowing of the airway below the glottis or apposing edges of the true vocal cords. In a broad definition, the term may be used to describe airway compromise in the regions of the larynx or the trachea or both.
F I, Catlin, R J, Smith
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Management of Subglottic Stenosis and Subglottic Stenosis in Systemic Disease
2012Subglottic stenosis (SGS) remains a difficult therapeutic problem for the interventional pulmonologist. It may be classified as congenital or acquired. Acquired causes frequently result from iatrogenic injuries. Further etiologies include tumors, infections, and rare diseases such as Wegener’s granulomatosis, relapsing polychondritis, and amyloidosis ...
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Subglottic Stenosis in Wegener's Granulomatosis
Chest, 1981A patient with treated Wegener's granulomatosis had an inflammatory subglottic laryngotracheitis during remission from systemic features of the disease. The lesion enlarged insidiously until a critical point of stenosis occurred in the subglottic region, leading to stridor.
J H, Lampman, R, Querubin, P, Kondapalli
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Management of Subglottic Stenosis in Children
Otolaryngology–Head and Neck Surgery, 1985We treated 21 children for subglottic stenosis at the Texas Children's Hospital from 1975 to 1983. Ages ranged from newborn to 14 years. Fifteen (71%) were younger than 13 months of age. Prolonged intubation was thought to be a primary contributing factor in 16 of 21 (76%). Nineteen (90%) required tracheotomy. Of these, 11 (58%) were decannulated, four
F I, Catlin, E M, Spankus
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Growth Factors in Subglottic Stenosis
Annals of Otology, Rhinology & Laryngology, 1996We sought to define the role of fibrogenic peptides in subglottic stenosis (SGS). Biopsy specimens were obtained from patients with stenosis following endotracheal intubation (group 1, n = 5, mean age 5), patients without a history of any precedent trauma, ie, idiopathic stenosis (group 2, n = 3, mean age 40), and those without stenosis (group 3, n = 3,
K A, Scioscia +3 more
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