Results 151 to 160 of about 22,657 (199)
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Mechanisms of supraventricular tachycardia

The American Journal of Cardiology, 1988
Programmed electrical stimulation of the heart in combination with intracardiac recordings has contributed a wealth of new information on the mechanisms and pathways of supraventricular tachycardia in humans. This knowledge has resulted in better treatment approaches to these patients.
H J, Wellens, P, Brugada
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Classification of supraventricular tachycardias

The American Journal of Cardiology, 1987
An ideal approach to classification of supraventricular arrhythmias would be based on exact knowledge of the pathophysiology and mechanism of the arrhythmia. Unfortunately, the mechanism may not be apparent from electrocardiographic data or indeed may not be known after extensive invasive and non-invasive studies.
G J, Klein   +3 more
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Supraventricular tachycardia in infancy

The American Journal of Cardiology, 1984
Our understanding of the mechanisms of cardiac arrhythmias in infants and children112 has advanced greatly since the classic monograph by Langendorf and Pick in 1954.3 Ambulatory and transtelephonic monitoring have accurately defined the occurrence of arrhythmias and invasive electrophysiologic studies have elucidated many of their mechanisms. Hubbard4
W E, Gaum, S, Kaplan
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Pathophysiology of supraventricular tachycardia

European Heart Journal, 1993
Supraventricular tachycardia is due to altered automaticity, triggered activity or most commonly, reentry. Atrial flutter is most frequently due to reentry with counterclockwise activation in the right atrium with the left atrium acting as a bystander.
C F, Shakespeare, M, Anderson, A J, Camm
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Persistent supraventricular tachycardia

The American Journal of Cardiology, 1965
Abstract A case of ectopic atrial tachycardia persisting for at least 11 years in a symptomless man of 37 is reported. This case points out that a high ventricular rate can be tolerated well for long periods of time if the cardiovascular system is undamaged and the ventricular rate is persistently slower than 200 beats per minute.
A, Dolara, L, Pozzi
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Introduction to Supraventricular Tachycardia

Cardiac Electrophysiology Clinics, 2010
Paroxysmal supraventricular tachycardia (PSVT) is a clinical syndrome characterized by a rapid tachycardia with an abrupt onset and termination cardiomyopathy. The three most common causes of PSVT are atrioventricular nodal reentrant tachycardia (50%-60%), atrioventricular reentrant tachycardia in patients with Wolff-Parkinson-White syndrome (25%-30%),
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Supraventricular tachycardia in children

The Indian Journal of Pediatrics, 2005
Several different mechanisms are responsible for paroxysmal supraventricular tachycardia in children. Different forms of tachycardia occur at different age. Atrio-ventricular reentry tachycardia results from the presence of congenital atrio-ventricular bypass tracts and is frequently encountered at all ages.
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Supraventricular tachycardia in children

Current Treatment Options in Cardiovascular Medicine, 2000
A reasonably precise and mechanistic diagnosis of the cause of supraventricular tachycardia (SVT) can be made using noninvasive tests such as an electrocardiogram, Holter monitoring, or cardiac event recorder and by determining the response to vagal maneuvers (or intravenous adenosine).
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Intrauterine supraventricular tachycardia

The Journal of Pediatrics, 1979
Six examples of intrauterine supraventricular tachycardia together with 31 previously reported cases are described and analyzed. Among the 37 infants, structural heart disease was present in only four (11%), three of whom died. Males comprised 68% of the group without identifiable heart disease or pre-excitation. Congestive heart failure was evident in
J W, Newburger, J F, Keane
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Adenosine and Supraventricular Tachycardia

New England Journal of Medicine, 1991
ADENOSINE is an endogenous nucleoside that is capable of causing atrioventricular nodal conduction block in humans.1 Although the use of intravenous adenosine to terminate supraventricular arrhythmias was first described in 1933,2 it is only in the past 8 years that adenosine has been investigated in detail3 and only in the past 18 months that it has ...
A J, Camm, C J, Garratt
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