Results 281 to 290 of about 70,302 (358)

Mitral Annulus Disjunction Causing Ventricular Tachyarrhythmias in a Young Lady With Palpitations, Cardiac Ablation Terminated the Complaints

open access: yesClinical Case Reports, Volume 13, Issue 4, April 2025.
ABSTRACT Mitral annulus disjunction (MAD) is a rare but significant cause of ventricular arrhythmias in young patients. Timely diagnosis using cardiac magnetic resonance imaging and targeted management with catheter ablation can effectively control arrhythmias, improve patient outcomes, and prevent severe complications such as sudden cardiac death.
Mohammad Hosein Nikoo   +3 more
wiley   +1 more source

An axis-specific mitral annuloplasty ring eliminates mitral regurgitation allowing mitral annular motion in an ovine model. [PDF]

open access: yesCommun Med (Lond)
Zhu Y   +26 more
europepmc   +1 more source

Cardiac remodelling in the era of the recommended four pillars heart failure medical therapy

open access: yesESC Heart Failure, Volume 12, Issue 2, Page 1029-1044, April 2025.
Abstract Cardiac remodelling is a key determinant of worse cardiovascular outcome in patients with heart failure (HF) and reduced ejection fraction (HFrEF). It affects both the left ventricle (LV) structure and function as well as the left atrium (LA) and the right ventricle (RV).
Giada Colombo   +7 more
wiley   +1 more source

Transendocardial injection of expanded autologous CD34+ cells after myocardial infarction: Design of the EXCELLENT trial

open access: yesESC Heart Failure, Volume 12, Issue 2, Page 1455-1463, April 2025.
Abstract Aims The extent of irreversible cardiomyocyte necrosis after acute myocardial infarction (AMI) is a major determinant of residual left ventricular (LV) function and clinical outcome. Cell therapy based on CD34+ cells has emerged as an option to help repair the myocardium and to improve outcomes.
Jerome Roncalli   +17 more
wiley   +1 more source

Metabolic abnormalities and reprogramming in cats with naturally occurring hypertrophic cardiomyopathy

open access: yesESC Heart Failure, Volume 12, Issue 2, Page 1256-1270, April 2025.
Abstract Background and aims The heart is a metabolic organ rich in mitochondria. The failing heart reprograms to utilize different energy substrates, which increase its oxygen consumption. These adaptive changes contribute to increased oxidative stress.
Qinghong Li   +12 more
wiley   +1 more source

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