Results 161 to 170 of about 49,177 (212)

Experimental tardive dyskinesia

Progress in Neuro-Psychopharmacology and Biological Psychiatry, 1982
In this work, we have attempted to reproduce dyskinesia similar to tardive dyskinesia by two methods. In the first experiment, we have administered to 6 macaca mulatta, haloperidol 0.25 mg/kg daily for six months. During that period we observed in all monkeys, after each dose: restlessness, akinesia and tremor.
P J, Bédard, R, Boucher, L, Larochelle
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Bromocriptine in Tardive Dyskinesia

Pharmacopsychiatry, 1982
Five schizophrenics and one alcoholic with tardive dyskinesia were given bromocriptine for two 8-week periods. The first trial was made during neuroleptic treatment and the second following a 3 weeks' wash-out of neuroleptics. When neuroleptics were given bromocriptine was essentially without effects, but after the neuroleptics had been discontinued ...
J E, Häggström, U, Andersson, L M, Gunne   +2 more
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TARDIVE DYSKINESIA IN THE ELDERLY

Age and Ageing, 1986
Four hundred and twenty-six elderly subjects were assessed for the presence of dyskinetic movements. Dyskinetic movements were present in 49 subjects (11.5%). Ninety-two per cent had orofacial movements, by far the commonest being chewing. Dyskinetic movements were present in 12.5% of women and 7.6% of men.
F M, Ramsay, P H, Millard
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Methylphenidate in Tardive Dyskinesia

American Journal of Psychiatry, 1973
Methylphenidate, an amphetamine-like central nervous system (CNS) stimulant used successfully in reversing acute neuroleptic-induced neurological symptoms, was given in a placebo-controlled study to 17 patients with tardive dyskinesia. On global ratings three subjects were improved, six showed an increase in symptoms, and eight showed no changes.
W E, Fann, J M, Davis, I C, Wilson
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Tardive Dyskinesia and Dementia

British Journal of Psychiatry, 1979
SummarySeventeen schizophrenic patients with tardive dyskinesia (TD) and 33 schizophrenics without tardive dyskinesia were examined by psychological tests of intellectual function and EMI scans were performed. The group as a whole were found to be demented and 31 out of 45 had abnormalities on the scan.
O O, Famuyiwa, D, Eccleston, A A, Donaldson, R F, Garside   +3 more
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TARDIVE DYSKINESIA AND ANTIHISTAMINES

Medical Journal of Australia, 1978
A case of tardive dyskinesia due to the prolonged administration of antihistamines is reported. Clinical features are discussed, as well as the alleviation of these by haloperidol.
C, Hale, T, Heins
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Smoking and tardive dyskinesia

Biological Psychiatry, 1987
Boisvert D, Chouinard G (1981): Rebound cardiac arrhythmia after withdrawal from imipramine: A case report. Am J Psych&y 138:985. Brown GM, Stancer HC, Moldofsky H, Harman J, Murphy YT, Gupta RM (1978): Withdrawal from long-term highdose desipramine therapy. Clinical and biological changes. Arch Gen Psychiatry 35:1261.
R L, Binder, H, Kazamatsuri, T, Nishimura, D E, McNiel   +3 more
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Quetiapine in tardive dyskinesia

International Journal of Psychiatry in Clinical Practice, 2002
Tardive dyskinesia (TD) is a potentially irreversible side-effect of antipsychotic medication. Some atypical antipsychotics, by virtue of their better side-effect profile, seem to have an ability to reverse TD. The importance of trying to treat TD has become more urgent in view of the medico-legal implications of Article 3 of the Human Rights Act, 1998
Suresh, Chari, Ak, Jainer, Andrew, Ashley-Smith, Maxine, Cleaver   +3 more
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Genetics of Tardive Dyskinesia

2011
Tardive dyskinesia (TD) is one of the most serious adverse side effects of antipsychotic drugs and is an important topic of pharmacogenetic studies. Since there is a genetic susceptibility for developing this adverse reaction, and given that it is hard to predict its development prior to or during the early period of medication, the genetic study of TD
Heon-Jeong, Lee, Seung-Gul, Kang
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Diagnosis of Tardive Dyskinesia

Clinical Neuropharmacology, 1983
The syndrome of tardive dyskinesia (TD) encompasses a series of abnormal involuntary movements associated with chronic exposure to neuroleptic agents. To establish the diagnosis with confidence, the history of prolonged medication must accompany the movements that characterize TD.
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