Results 11 to 20 of about 1,415,735 (309)

Temporomandibular joint disorders (Review Article) [PDF]

open access: yesSulaimani Dental Journal, 2016
Temporomandibular joint (TMJ) is a sensitive and highly mobile joint. Temporomandibular disorders (TMDs) have been considered as a common orofacial pain condition.
Shanaz Gaphor
doaj   +3 more sources

TGFBI Maintains Fibrocartilage Homeostasis by Restraining O-GlcNAcylation to Inactivate and Destabilize ADAMTS16 in Temporomandibular Joint Osteoarthritis. [PDF]

open access: yesAdv Sci (Weinh)
TGFBI deficiency elevates O‐GlcNAcylation, promoting the transcriptional activation of ADAMTS16 and OGT‐mediated post‐translational modification of ADAMTS16 at Ser1170. This leads to its stabilization and accumulation, driving chondrocyte metabolic disorders and senescent phenotypes, ultimately resulting in fibrocartilage dysfunction and TMJOA ...
Liu X   +12 more
europepmc   +2 more sources

A study of the influence of low intensity laser therapy on painful temporomandibular disorders [PDF]

open access: yes, 1998
PhDAn analgesic effect is one of the reported results of Low Intensity Laser Therapy (LILT). Even though this therapy has been extensively used to treat musculoskeletal injury, pain and inflammation, there is still a lack of research concerning the ...
Sattayut, Sajee
core   +4 more sources

Assessment of five oblique radiographic projections of the canine temporomandibular joint [PDF]

open access: yes, 2012
Investigation of temporomandibular joint (TMJ) disease requires a clear diagnostic image, which can be challenging to obtain using conventional radiography.
Hammond, G., King, A., LaPaglia, J.
core   +1 more source

Arthrotomography of the temporomandibular joint [PDF]

open access: yesAmerican Journal of Roentgenology, 1980
A series of 104 arthrotomographic examinations of the temporomandibular joint in 89 patients is described. Those patients with symptoms referable to the temporomandibular joint and not responsive to conservative methods of therapy were selected. The mean patient age was 29 years with a 3:1 female to male ratio.
R W, Katzberg   +5 more
openaire   +2 more sources

Temporomandibular Joint Pathology and Its Indication in Clinical Orthodontics [PDF]

open access: yes, 2017
Temporomandibular joint (TMJ) pathology has been an area of study in dentistry specifically a research interest in clinical orthodontics in which treatment option has been a dilemma for practitioners.
Ho, KCJ, Rabie, ABM, Li, Q
core   +1 more source

Temporomandibular joint dysfunctions in the context of psychological disorders among pediatric patients

open access: yesJournal of Education, Health and Sport, 2019
Introduction: There are many factors that affect temporomandibular joint dysfunction. These may include, among others: structural and postural disorders, bruxism, and occlusive abnormalities.
Agnieszka Zuszek   +3 more
doaj   +1 more source

Temporomandibular disorders in patients with craniocervical dystonia

open access: yesArquivos de Neuro-Psiquiatria, 2011
Temporomandibular disorders are a set of musculoskeletal dysfunctions within the masticatory system, with multiple etiologies. OBJECTIVE: Since craniocervical dystonia can involve the same neuromuscular structure as the temporomandibular joint, we sought
André L. Costa   +3 more
doaj   +1 more source

Three-dimensional retrospective comparison of disc repositioning, condylar remodelling, and mandibular symmetry following traditional versus digital articulation techniques in patients with temporomandibular disorders

open access: yesBMC Oral Health
Background With advancements in computer technology, digital mandibular repositioning techniques are increasingly utilised in the management of temporomandibular disorders (TMDs). This study aimed to compare the differences in joint structure restoration
Jinyi Zhu   +5 more
doaj   +1 more source

BAP31 Drives Cartilage Calcification through Disruption of Autophagosome–Lysosome Fusion in Osteoarthritis

open access: yesAdvanced Science, EarlyView.
Mechanical stress activates BAP31 in chondrocytes. BAP31 competes with ATG14 for binding to STX17, disrupting the STX17–ATG14 complex required for autophagosome–lysosome fusion. The resultant autophagic flux blockade drives the generation of autophagy‐derived exosomes, which mediate pathological cartilage calcification in OA. Chondrocyte‐targeted BAP31
Zhi‐hua Xu   +13 more
wiley   +1 more source

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