Results 361 to 370 of about 432,090 (383)
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Seminars in Fetal and Neonatal Medicine, 2008
Fetal thrombocytopenia is most often caused by maternal alloantibodies against fetal platelets crossing the placenta and resulting in platelet destruction. This condition, known as fetal and neonatal alloimmune thrombocytopenia, is usually detected after the birth of a symptomatic child who shows signs of bleeding in the skin or in the brain.
L, Porcelijn +2 more
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Fetal thrombocytopenia is most often caused by maternal alloantibodies against fetal platelets crossing the placenta and resulting in platelet destruction. This condition, known as fetal and neonatal alloimmune thrombocytopenia, is usually detected after the birth of a symptomatic child who shows signs of bleeding in the skin or in the brain.
L, Porcelijn +2 more
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Current Opinion in Hematology, 1995
Congenital thrombocytopenias are rare bleeding disorders but must be included in the differential diagnosis when investigating a young infant with chronic thrombocytopenia. Several of these syndromes have associated defects of immune, renal, or skeletal systems in addition to thrombocytopenia.
I, Warrier, J M, Lusher
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Congenital thrombocytopenias are rare bleeding disorders but must be included in the differential diagnosis when investigating a young infant with chronic thrombocytopenia. Several of these syndromes have associated defects of immune, renal, or skeletal systems in addition to thrombocytopenia.
I, Warrier, J M, Lusher
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Hospital Practice, 1983
The common denominator of this group of disorders is bleeding due to antibody-mediated platelet destruction. The variations in clinical manifestation--ranging from mild to fatal--reflect the diverse origins of the troublesome antibodies: transplacental, exogenous, and autogenous.
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The common denominator of this group of disorders is bleeding due to antibody-mediated platelet destruction. The variations in clinical manifestation--ranging from mild to fatal--reflect the diverse origins of the troublesome antibodies: transplacental, exogenous, and autogenous.
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Journal of Pediatric Hematology/Oncology, 2003
Alloimmunization against platelet antigens may take place during pregnancy and following transfusion or transplantation. The resulting antibodies bind to the target platelet antigens and may lead to a rapid sequestration of incompatible platelets.
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Alloimmunization against platelet antigens may take place during pregnancy and following transfusion or transplantation. The resulting antibodies bind to the target platelet antigens and may lead to a rapid sequestration of incompatible platelets.
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Paraneoplastic Thrombocytopenia
The American Journal of Medicine, 2021Vasilios Kalas +5 more
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Heparin-Induced Thrombocytopenia
Australian and New Zealand Journal of Medicine, 1988Abstract:Thrombocytopenia is a common adverse effect of heparin therapy. Two types of heparinlinduced thrombocytopenia (HIT) are observed clinically ‐ an early onset mild thrombocytopenia (Type I) in which the patients remain asymptomatic and a delayed onset severe thrombocytopenia (Type II).
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Seminars in Hematology, 2006
Thrombocytopenia in the newborn period can signify an inherited platelet disorder. Congenital amegakaryocytic thrombocytopenia (CAMT) and thrombocytopenia with absent radii (TAR) share features of isolated thrombocytopenia, reduced or absent marrow megakaryocytes, impaired responsiveness to thrombopoietin (TPO), and high plasma TPO levels.
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Thrombocytopenia in the newborn period can signify an inherited platelet disorder. Congenital amegakaryocytic thrombocytopenia (CAMT) and thrombocytopenia with absent radii (TAR) share features of isolated thrombocytopenia, reduced or absent marrow megakaryocytes, impaired responsiveness to thrombopoietin (TPO), and high plasma TPO levels.
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New England Journal of Medicine, 2020
Virinder, Nohria, A Christopher, Macie
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Virinder, Nohria, A Christopher, Macie
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Heparin-Induced Thrombocytopenia
Annual Review of Medicine, 1989Heparin-induced thrombocytopenia (HIT) is an immune-mediated adverse drug effect that is characterized by platelet activation, hypercoagulability, and a resulting increased risk for thrombosis, both venous and arterial. This disorder is autoimmune-like, because the target antigen is a multimolecular complex of the "self" protein, platelet factor 4, and
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Clinical Obstetrics and Gynecology, 1999
N, Shehata, R, Burrows, J G, Kelton
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N, Shehata, R, Burrows, J G, Kelton
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