Results 151 to 160 of about 11,815 (206)

Timolol

open access: yes, 2007
Timolol is a non-selective, beta adrenoceptor antagonist at beta-1 and beta-2 adrenoceptors. It lacks the partial agonist and local anesthetic properties that some drugs in this class have. It is most commonly used to treat glaucoma and cardiovascular diseases. Timolol is moderately lipophilic. Absorption of timolol after topical application to the eye
Donald Hoover, Hoover, Donald
openaire   +3 more sources

Timolol and Glaucoma

Archives of Ophthalmology, 1981
To the Editor. —There is increasing evidence of systemic side effects secondary to topical ocular administration of timolol maleate for the treatment of glaucoma. 1,2 These systemic side effects are essentially the same as those seen from systemically administered β-blockers.
E M, Van Buskirk, F T, Fraunfelder
openaire   +2 more sources

Travoprost/Timolol

Drugs & Aging, 2006
Travoprost 0.004%/timolol 0.5% fixed combination (travoprost/timolol) is a once-daily eyedrops solution comprising the prostaglandin F(2alpha) analogue travoprost and the beta-adrenoceptor antagonist timolol. It is indicated for the treatment of patients with open-angle glaucoma or ocular hypertension who are insufficiently responsive to topical beta ...
Sheridan M, Hoy   +2 more
openaire   +2 more sources

Pharmacogenetics of timolol

Vestnik oftal'mologii, 2019
In recent years, β-adrenergic blockers have become the first choice drugs for glaucoma treatment. Timolol holds the main position among them, being a part of most combined antiglaucoma preparations. The use of timolol maleate in clinical practice may be accompanied by severe side effects affecting different organs and systems.
L K, Moshetova   +3 more
openaire   +2 more sources

Timolol and cornea

Acta Ophthalmologica, 1986
Abstract The influence of timolol on the corneal epithelium and endothelium was studied in organ culture. The cell morphology was not influenced by drug concentrations ≦ 690 μg/ml. Cell death was observed at 6900 μg/ml. Endothelial cell proliferation after an experimental injury was slowed down at a drug concentration of 690 μg/ml.
E G, Olsen, M, Davanger
openaire   +2 more sources

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