Results 141 to 150 of about 4,605 (177)
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Journal of Urology, 1985
Triamterene therapy is an unusual cause of nephrolithiasis and, when this agent is found in a stone, generally it is deposited in minor amounts. We report a renal calculus consisting mostly of triamterene and its 2 major metabolites in a patient taking a triamterene-containing drug, and discuss some implications.
E S, Dickstein, W D, Loeser
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Triamterene therapy is an unusual cause of nephrolithiasis and, when this agent is found in a stone, generally it is deposited in minor amounts. We report a renal calculus consisting mostly of triamterene and its 2 major metabolites in a patient taking a triamterene-containing drug, and discuss some implications.
E S, Dickstein, W D, Loeser
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Military Medicine, 1989
A 59-year-old white male presented with a triamterene renal stone and pyelonephritis, requiring percutaneous lithotomy. A combination of hydrochlorothiazide and triamterene had been previously prescribed despite no history of hypokalemia. The indications for potassium replacement therapy and risks of triamterene nephrolithiasis are reviewed.
D P, Dooley, M E, Callsen, J A, Geiling
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A 59-year-old white male presented with a triamterene renal stone and pyelonephritis, requiring percutaneous lithotomy. A combination of hydrochlorothiazide and triamterene had been previously prescribed despite no history of hypokalemia. The indications for potassium replacement therapy and risks of triamterene nephrolithiasis are reviewed.
D P, Dooley, M E, Callsen, J A, Geiling
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Drug and Therapeutics Bulletin, 1964
Triamterene (2, 4, 7-triamino-6-phenyl pteridine, Dytac - SKF) is a new oral diuretic. It increases the urinary excretion of sodium, chloride and bicarbonate, but decreases the urinary loss of potassium. The diuretic effect of triamterene depends neither on carbonic anhydrase nor on antagonism of aldosterone, but appears to be due to a direct action of
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Triamterene (2, 4, 7-triamino-6-phenyl pteridine, Dytac - SKF) is a new oral diuretic. It increases the urinary excretion of sodium, chloride and bicarbonate, but decreases the urinary loss of potassium. The diuretic effect of triamterene depends neither on carbonic anhydrase nor on antagonism of aldosterone, but appears to be due to a direct action of
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Triamterene Crystalline Nephropathy
American Journal of Kidney Diseases, 2014Medications can cause a tubulointerstitial insult leading to acute kidney injury through multiple mechanisms. Acute tubular injury, a dose-dependent process, occurs due to direct toxicity on tubular cells. Acute interstitial nephritis characterized by interstitial inflammation and tubulitis develops from drugs that incite an allergic reaction.
Samih H, Nasr +3 more
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Triamterene-Induced Nephrolithiasis
Annals of Internal Medicine, 1979Excerpt A patient passed kidney stones composed entirely of a mixture of triamterene and a metabolic product of triamterene.
B, Ettinger +3 more
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Pharmacokinetics of Triamterene
Clinical and Experimental Hypertension. Part A: Theory and Practice, 1983The knowledge about the pharmacokinetics of triamterene (TA) was limited until recently. The metabolic pathway of TA is the formation of p-hydroxytriamterene (OH-TA), which is subsequently conjugated with active sulfate to form p-hydroxytriamterene sulfuric acid ester (OH-TA-ester). The phase-II-metabolite is surprisingly pharmacologically active.
E, Mutschler +4 more
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