Results 271 to 280 of about 109,157 (295)
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Fabrication of Protein Tubules: Immobilization of Proteins on Peptide Tubules
The Journal of Physical Chemistry B, 2001Peptide tubules, self-assemblies of bis(N-α-amido-glycylglycine)-1,7-heptane dicarboxylate, were functionalized by poly(l-lysine), d-biotin, avidin, and albumin bovine. Proteins were covalently immobilized on the peptide tubules via NHS ester intermediates, while amide groups of the peptide tubules were also capable of intercalating proteins via ...
Gary E. Douberly +3 more
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Melting of Phospholipid Tubules
Physical Review Letters, 1996The melting of lipid-based microcylinders (tubules) has been investigated for systems with single and multiple bilayer walls using high field, magnetic birefringence, and precision microcalorimetry. The pretransitional behavior of both the magnetic birefringence and the specific heat is very different in tubules with a single bilayer wall from that of ...
George Nounesis +8 more
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Nanocapillarity in fullerene tubules
Physical Review Letters, 1992Fullerene tubules are shown to be highly polarizable ``molecular straws'' capable of ingesting dipolar molecules. Local-density-functional calculations on HF molecules within a finite-length tubule, of size 144 atoms, demonstrate this effect. The energy of incarceration is several times the thermal ambient at room temperature.
Mark R. Pederson, Jeremy Q. Broughton
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Hospital Practice, 1982
Genetic defects in tubule function, although relatively rare, have served as models for the study of the more common acquired renal tubular disorders. Defects in reabsorption or secretion or in hormone production or action are primarily involved. An exact physiologic mechanism has been delineated for some of them, leading to effective management of the
Ralph A. DeFronzo, S. O. Thier
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Genetic defects in tubule function, although relatively rare, have served as models for the study of the more common acquired renal tubular disorders. Defects in reabsorption or secretion or in hormone production or action are primarily involved. An exact physiologic mechanism has been delineated for some of them, leading to effective management of the
Ralph A. DeFronzo, S. O. Thier
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Role of Transverse Tubules (T-Tubules) in Muscle Glucose Transport
1998The first data to demonstrate glucose transporter translocation in muscle used membranes enriched in sarcolemma because it was assumed that this was the equivalent of the cell membrane of adipocytes. We studied translocation in intact human muscle using immunogold labeling of the GLUT4 transporter but found very little labeling on the sarcolemma.
Ronald W. Dudek, G. Lynis Dohm
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Magnetization of graphene tubules
Physical Review B, 1995Magnetization (M) comes from both the persistent currents and the spin polarization. The spin-B interaction is important in a graphene tubule, because it makes the one-dimensional subband with the divergent density of states capable of crossing the Fermi level (${\mathit{E}}_{\mathit{F}}$=0 eV).
Min-Fa Lin, Kenneth W. K. Shung
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Extracellular Tubules in Semen
Journal of Andrology, 1990Tubules were observed in the semen of two patients, one of whom was fertile. The tubules measured 75 to 90 nm in diameter and were extracellular. However, they frequently were found in amorphous material reminiscent of cell debris and in association with cells, most of which were immature.
Georg Schieferstein, Hartwig Wolburg
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1994
The morphology of the proximal tubule cells was covered briefly in Chapter 2, but will be considered in more detail here. The proximal tubule is divisible into the convoluted portion, or pars convoluta, which begins immediately behind the glomerulus, and the straight portion, or pars recta, which passes into the medulla to become the loop of Henle. The
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The morphology of the proximal tubule cells was covered briefly in Chapter 2, but will be considered in more detail here. The proximal tubule is divisible into the convoluted portion, or pars convoluta, which begins immediately behind the glomerulus, and the straight portion, or pars recta, which passes into the medulla to become the loop of Henle. The
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1995
Tubulitis in the kidney is defined as the presence of inflammatory cells in the tubular wall (OOi et al. 1975). It is the hallmark of tubulointerstitial nephritis (OLsen etal. 1986) and one of the important lesions in acute renal allograft rejection (SOlez et al. 1993). However, it may occur in other renal diseases as well, e.g., glomerulonephritis and
B. Iványi, S. Olsen
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Tubulitis in the kidney is defined as the presence of inflammatory cells in the tubular wall (OOi et al. 1975). It is the hallmark of tubulointerstitial nephritis (OLsen etal. 1986) and one of the important lesions in acute renal allograft rejection (SOlez et al. 1993). However, it may occur in other renal diseases as well, e.g., glomerulonephritis and
B. Iványi, S. Olsen
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2003
Publisher Summary This chapter focuses on the various diseases involving renal tubule, which plays a key role in the body's regulation of fluid, electrolyte, and acid–base balance. The vast majority of water and electrolytes in the glomerular ultrafiltrate are reabsorbed along the tubule by specialized transporters and channels, specifically localized
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Publisher Summary This chapter focuses on the various diseases involving renal tubule, which plays a key role in the body's regulation of fluid, electrolyte, and acid–base balance. The vast majority of water and electrolytes in the glomerular ultrafiltrate are reabsorbed along the tubule by specialized transporters and channels, specifically localized
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