Results 11 to 20 of about 6,484,256 (294)
On weak-type (1, 1) for averaging type operators
Journal of Functional Analysis, 2023 It is known that, due to the fact that $L^{1, \infty}$ is not a Banach space, if $(T_j)_j$ is a sequence of bounded operators so that $$ T_j:L^1\longrightarrow L^{1, \infty}, $$ with norm less than or equal to $||T_j||$ and $\sum_j ||T_j||<\infty$, nothing can be said about the operator $T=\sum_j T_j$.
Sergi Baena-Miret, María J. Carro
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Neurofibromatosis type 1 [PDF]
Journal of the American Academy of Dermatology, 2009 Neurofibromatosis type 1 (NF1) is an autosomal dominant, multisystem disorder affecting approximately 1 in 3500 people. Significant advances in the understanding of the pathophysiology of NF1 have been made in the last decade. While no medical therapies for NF1 are currently available, trials are ongoing to discover and test medical treatments for the ...
Kevin P. Boyd, Bruce R. Korf, Amy Theos
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Clinical Chemistry, 1999 AbstractType 1 (insulin-dependent) diabetes occurs worldwide and can appear at any age. The genetic susceptibility is strongly associated with HLA-DQ and DR on chromosome 6, but genetic factors on other chromosomes such as the insulin gene on chromosome 11 and the cytotoxic T-lymphocyte antigen gene on chromosome 2 may modulate disease risk.
Åke Lernmark, Shehab Alshiekh
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PDE12 in type 1 diabetes [PDF]
Scientific Reports, 2022 AbstractType 1 diabetes (T1D) incidence is increased after COVID-19 infection in children under 18 years of age. Interferon-α-activated oligoadenylate synthetase and downstream RNAseL activation degrade pathogen RNA, but can also damage host RNA when RNAseL activity is poorly regulated.
Tekin, Hasim +6 more
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Neurofibromatosis type 1 [PDF]
QJM: An International Journal of Medicine, 2018 The neurofibromatoses are a group of three heterogeneous disorders that include neurofibromatosis type 1 (NF1), neurofibromatosis type 2 (NF2), and schwannomatosis. NF1 is the most common of these three conditions, and represents one of the most frequently diagnosed cancer predisposition disorders involving the nervous system.
Tsung-Han Ho +4 more
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The Lancet, 2014 Over the past decade, knowledge of the pathogenesis and natural history of type 1 diabetes has grown substantially, particularly with regard to disease prediction and heterogeneity, pancreatic pathology, and epidemiology. Technological improvements in insulin pumps and continuous glucose monitors help patients with type 1 diabetes manage the challenge ...
George S. Eisenbarth +2 more
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The Lancet, 2018 Type 1 diabetes is a chronic autoimmune disease characterised by insulin deficiency and resultant hyperglycaemia. Knowledge of type 1 diabetes has rapidly increased over the past 25 years, resulting in a broad understanding about many aspects of the disease, including its genetics, epidemiology, immune and β-cell phenotypes, and disease burden ...
DiMeglio, Linda A. +2 more
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The Lancet, 2023 Type 1 diabetes is a chronic disease caused by autoimmune destruction of pancreatic β cells. Individuals with type 1 diabetes are reliant on insulin for survival. Despite enhanced knowledge related to the pathophysiology of the disease, including interactions between genetic, immune, and environmental contributions, and major strides in treatment and ...
Quattrin, Teresa +2 more
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Lung India, 2019 Although being the second most common cause of disabling daytime sleepiness in the world, narcolepsy in India is seldom diagnosed. So far, only two cases had been reported. This is the case of narcolepsy in a 15-year-old female student struggling in her scholastic pursuit because of the disease.
Rajesh Swarnakar, Akshay Deotare
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Inflammasomes and Type 1 Diabetes [PDF]
Frontiers in Immunology, 2021 Microbiota have been identified as an important modulator of susceptibility in the development of Type 1 diabetes in both animal models and humans. Collectively these studies highlight the association of the microbiota composition with genetic risk, islet autoantibody development and modulation of the immune responses.
F. Susan Wong, James A. Pearson, Li Wen
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