Results 51 to 60 of about 51,066 (193)

Unstable Angina [PDF]

open access: yesCirculation, 1998
Unstable angina results from an imbalance between myocardial oxygen supply and demand. Probably the most common cause is reduced myocardial perfusion resulting from a nonocclusive thrombus on a fissured or eroded atherosclerotic plaque that often had caused only mild to moderate obstruction previously.3 Nonocclusive thrombi in patients with unstable ...
openaire   +2 more sources

Association of the 894G>T polymorphism of the endothelial constitutive nitric oxide synthase gene with unstable angina

open access: yesBrazilian Journal of Medical and Biological Research, 2007
The 894G>T polymorphism of the endothelial constitutive nitric oxide synthase gene consists of the substitution of a guanine base by a thymine at the 894th nucleotide of the gene. An association of this polymorphism with acute coronary syndromes has been
G.R. Iturry-Yamamoto   +4 more
doaj  

Microaxial Flow Pump Support as a Bridge to Transaxillary TAVI‐In‐TAVI in Cardiogenic Shock

open access: yesClinical Case Reports, Volume 14, Issue 8, August 2026.
ABSTRACT A 79‐year‐old man presented with cardiogenic shock due to severe degeneration of a prior transcatheter aortic valve. Temporary mechanical circulatory support with a transvalvular microaxial flow pump was initiated, resulting in rapid stabilization.
Matteo Orlandi   +9 more
wiley   +1 more source

A Therapeutic Dilemma: Catastrophic ST‐Elevation Myocardial Infarction Following Antiplatelet Therapy Interruption in a Patient With JAK2‐Positive Polycythemia Vera

open access: yesClinical Case Reports, Volume 14, Issue 8, August 2026.
Premature interruption of dual antiplatelet therapy in a patient with polycythemia vera and clinically suspected acquired von Willebrand syndrome triggered catastrophic recurrent stent thrombosis, whereas perioperative bridging with eptifibatide enabled safe surgery. ABSTRACT Polycythemia vera is associated with a thrombotic–hemorrhagic paradox.
Behrouz zarei   +2 more
wiley   +1 more source

Angina Pectoris - A general practice review: Part I

open access: yesSouth African Family Practice, 1989
No abstract available.
E.L. Murray
doaj   +1 more source

The Association between Hypoperfused Myocardium and Systolic Function in Patients With AMI Undergoing Complete Revascularization

open access: yesClinical Cardiology, Volume 49, Issue 8, August 2026.
Hypoperfused myocardium was detected in portions of acute myocardial infarction patients undergoing complete revascularization by hybrid positron emission tomography/magnetic resonance at 2 months. Patients with hypoperfused myocardium (Group A) had higher hs‐CRP and lower T2 values than those without (Group B). The reversal of myocardial perfusion was
Jin Si   +11 more
wiley   +1 more source

EXERCISE TESTS IN UNSTABLE ANGINA SUSPECTS

open access: yesТерапевтический архив, 2012
The review is devoted to exercise tests (ET) potential in patients with different forms of coronary heart disease (CHD) exacerbation and suspected unstable angina.
M N Alekhin, N V Eremina, N F Radova
doaj  

Ativação plaquetária em formas clínicas distintas da doença arterial coronariana (papel da P-selectina e de outros marcadores nas anginas estável e instável) Platelet activation in different clinical forms of the coronary artery disease (roll of P-selectin and others platelet markers in the stable and unstable angina)

open access: yesArquivos Brasileiros de Cardiologia, 2006
OBJETIVO: Os marcadores da ativação plaquetária em geral se apresentam elevados na doença arterial coronariana. Desse modo, procuramos identificar a presença e as potenciais associações de diferentes marcadores da ativação plaquetária. MÉTODOS: Estudamos
Margareth L. Venturinelli   +6 more
doaj   +1 more source

Extracellular Vesicle cystatin c is associated with unstable angina in troponin negative patients with acute chest pain.

open access: yesPLoS ONE, 2020
BackgroundDespite the use of high-sensitive cardiac troponin there remains a group of high-sensitive cardiac troponin negative patients with unstable angina with a non-neglectable risk for future adverse cardiovascular events, emphasising the need for ...
Mirthe Dekker   +10 more
doaj   +1 more source

Impact of CYP2C19 and CYP3A4 Inhibitor Use on Clopidogrel Clinical Effectiveness in CYP2C19 Genotyped Patients Undergoing Percutaneous Coronary Intervention

open access: yesClinical Pharmacology &Therapeutics, Volume 120, Issue 2, Page 531-541, August 2026.
CYP2C19 and CYP3A4 contribute to clopidogrel bioactivation. CYP2C19 no‐function alleles diminish clopidogrel's antiplatelet effects and clinical effectiveness. Coadministration of either a CYP2C19 or a CYP3A4 inhibitor may also reduce clopidogrel's antiplatelet effects and lead to phenoconversion in patients without a CYP2C19 no‐function allele (normal/
Danwei Shao   +8 more
wiley   +1 more source

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