Results 261 to 270 of about 5,591,095 (382)
Non‐small cell lung cancer targeted treatment is limited to a few known genetic alterations, with few alternatives in advanced treatment lines. To direct treatment decisions by drug sensitivity testing (DST), this study compared several methods for tumor cell isolation from malignant effusions, pointing to repeated CD45+ cell depletion for effective ...
Navit Mooshayef+10 more
wiley +1 more source
STAR-RIS enabled covert integration of sensing communication and over-the-air computing with analysis and optimization. [PDF]
Zhang M, Wang C, Ren T, Li J.
europepmc +1 more source
Human cytomegalovirus infection is common in normal prostate epithelium, prostate tumor tissue, and prostate cancer cell lines. CMV promotes cell survival, proliferation, and androgen receptor signaling. Anti‐CMV pharmaceutical compounds in clinical use inhibited cell expansion in prostate cancer models in vitro and in vivo, motivating investigation ...
Johanna Classon+13 more
wiley +1 more source
A burden of proof study of the effects of exposure to high fasting plasma glucose on the risk of seven types of cancer. [PDF]
Teixeira PP+14 more
europepmc +1 more source
Carcinoma‐associated fibroblasts (CAFs) in tumors influence cancer progression. We identified endoglin (ENG) as a key factor in TGF‐β signaling in myofibroblastic CAFs (myCAFs), linked to poor breast cancer outcomes. Inhibiting ENG on myCAFs suppressed the TGF‐β‐Smad2/3 pathway, reducing primary tumor growth and metastasis.
Shoki Okubo+11 more
wiley +1 more source
Modeling the importance of life exposure factors on memory performance in diverse older adults: A machine learning approach. [PDF]
Fletcher E+9 more
europepmc +1 more source
Spectral Classification of the Edmondson-Rogers Variable Star [PDF]
Roscoe F. Sanford
openalex +1 more source
IX. Observations of a new variable star
J. Goodricke
semanticscholar +1 more source
In luminal (ER+) breast carcinoma (BC), miRNA profiling identified miR‐195‐5p as a key regulator of proliferation that targets CHEK1, CDC25A, and CCNE1. High CHEK1 expression correlates with worse relapse‐free survival after chemotherapy, especially in patients with luminal A subtype.
Veronika Boušková+14 more
wiley +1 more source