Results 351 to 360 of about 204,625 (386)
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The American Journal of Medicine, 1986
Vasodilators are a group of drugs with various degrees of arteriolar or venous dilatation used in the treatment of congestive heart failure when symptoms persist after digoxin and diuretic therapy. Nitrates and captopril provide the most consistent improvement in symptoms. Reduced mortality rates in congestive heart failure with vasodilator therapy has
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Vasodilators are a group of drugs with various degrees of arteriolar or venous dilatation used in the treatment of congestive heart failure when symptoms persist after digoxin and diuretic therapy. Nitrates and captopril provide the most consistent improvement in symptoms. Reduced mortality rates in congestive heart failure with vasodilator therapy has
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Intercellular signal for vasodilation [PDF]
Vascular Biology Vasoconstriction must be balanced with vasodilation in the arterioles that supply tissues with blood. The second messenger IP3 has been thought to be the signal that passes from smooth muscle cells through gap junctions to endothelial cells to trigger vasodilation. However, Garland et al.
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Vasodilating Mechanisms of Testosterone
Experimental and Clinical Endocrinology & Diabetes, 2007The increased incidence of cardiovascular disease in man compared with premenopausal women suggests an unfavourable effect of male sex hormone testosterone on the cardiovascular system. However, numerous clinical and epidemiological studies reported a controversial relationship between testosterone and cardiovascular disease. Furthermore, an increasing
Melik Seyrek, Oguzhan Yildiz
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Hyperhomocyst(e)inemia is associated with impaired endothelium-dependent vasodilation in humans.
Circulation, 1997BACKGROUND Hyperhomocyst(e)inemia is a risk factor for atherosclerosis and is prevalent in the elderly. The objective of this study was to determine whether hyperhomocyst(e)inemia is associated with impaired endothelium-dependent vasodilation in humans.
A. Tawakol+4 more
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Nifedipine: A Novel Vasodilator
Drugs, 2006Nifedipine was synthesized by Bayer Germany in 1966 and considered for clinical use as a coronary vasodilator in patients with angina pectoris. Japanese investigators played a great part in the pre-clinical and clinical development of nifedipine. Professor Hashimoto demonstrated that nifedipine could increase coronary blood flow even in extremely low ...
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Vasodilation in skeletal muscle
American Journal of Physiology-Legacy Content, 1963Blood flow was studied in dog hind-limb muscle isolated except for femoral and sciatic nerves. At constant perfusion pressure, elevation of aortic pressure produced by blood transfusion or intravenous epinephrine administration elicited a three- to fourfold increase in blood flow in perfused muscle.
Richard D. Jones, Robert M. Berne
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Sympathetic Vasodilator Outflow
Physiological Reviews, 1954Abstract : Excitation of the sympathetic vasodilator outflow by hypothalamic and medullary stimulation causes a concomitant increase of cardiac contractile force and of skeletal muscle blood flow indicating those effects being integral parts of the reaction pattern elicited by intracerebral activation of the sympathetic vasodilator system.
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Sympathomimetics, Inotropics, and Vasodilators
AACN Advanced Critical Care, 1992Management of the critically ill patient often necessitates the use of multiple inotropic and vasoactive medications. This article offers a concise, clinical reference to guide the practicing clinician in the use of these agents. The focus is on parenteral agents used for acute rather than chronic management. Physiologic concepts basic to understanding
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Vasodilation for Vasospastic Hypertension
New England Journal of Medicine, 1973Accelerated or malignant hypertension is a progressive vasospastic disease characterized by rapidly increasing peripheral vascular resistance. Arterial pressure must be quickly and effectively reduced to halt arteriolar deterioration and to prevent advancing renal damage, hemorrhagic complications, or hypertensive encephalopathy.
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