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Intracranial Aneurysm Rupture, Vasospasm, and Infarction
Archives of Neurology, 1964During a recent study of the relationship between spontaneous rupture of saccular intracranial aneurysms and cerebral infarction, a preliminary comparison with angiographic reports of cerebral vasospasm was made. 1 Infarcts were found in the brains of 65% of patients in whose angiograms vasospasm had been noted, while 82% of patients for whom vasospasm
S A, SCHNECK, I I, KRICHEFF
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Significance of vasospasm in the treatment of ruptured intracranial aneurysms
Journal of Neurosurgery, 1977✓ The authors have analyzed a total of 96 consecutive cases in which vasospasm followed subarachnoid hemorrhage (SAH). The SAH was caused by ruptured intracranial aneurysm or developed after aneurysm surgery. Usually at least 4 days elapsed between SAH and the onset of vasospasm. Vasospasm subsided an average of 2 weeks after onset. Of 68 patients with
I, Saito, Y, Ueda, K, Sano
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Transluminal angioplasty for treatment of intracranial arterial vasospasm
Journal of Neurosurgery, 1989✓ Percutaneous transluminal angioplasty for treatment of intracerebral arterial vasospasm is now being performed in selected cases. Thirty-six vascular territories in 13 patients, ranging in age from 15 to 73 years, have been treated with a new silicone microballoon device.
R T, Higashida +6 more
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Vasospasm With Subarachnoid Hemorrhage From Intracranial Aneurysm
Archives of Surgery, 1965VASOSPASM accompanying aneurysm of the intracranial portion of the internal carotid artery or its branches or other cerebral vessels may complicate the clinical picture and also render surgical management of the aneurysm more difficult. It may occur whether or not the aneurysm has bled but is more common after subarachnoid hemorrhage from leakage or ...
C D, HAWKES, W S, OGLE
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Use of Extracranial-Intracranial Bypass in the Management of Symptomatic Vasospasm
Neurosurgery, 1986Abstract Delayed ischemic deficits from vasospasm after subarachnoid hemorrhage remain a major source of death and disability to patients surviving subarachnoid hemorrhage. Ideal treatment for this condition would prevent or reverse spasm in major subarachnoid vessels. This goal remains elusive.
H, Batjer, D, Samson
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