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2001
Delayed release of hemoglobin from the subarachnoid clot in the vicinity of conductive arteries leads to cerebral vasospasm after SAH. The mechanism of vasospasm however remains unclear. The one thousand times higher affinity of ferrous heme for nitric oxide, a potent vasodilator of cerebral vessels, than to oxygen, has led to the concept that ...
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Delayed release of hemoglobin from the subarachnoid clot in the vicinity of conductive arteries leads to cerebral vasospasm after SAH. The mechanism of vasospasm however remains unclear. The one thousand times higher affinity of ferrous heme for nitric oxide, a potent vasodilator of cerebral vessels, than to oxygen, has led to the concept that ...
R M, Pluta +5 more
openaire +2 more sources
Management of cerebral vasospasm
Neurosurgical Review, 2006Cerebral vasospasm is delayed narrowing of the large arteries of the circle of Willis occurring 4 to 14 days after aneurysmal subarachnoid hemorrhage (SAH). It is but one cause of delayed deterioration after SAH but, in general, is the most important potentially treatable cause of morbidity and mortality after SAH.
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