Results 291 to 300 of about 200,200 (400)

Transparavertebral Occlusion of a Large Venovenous Collateral in a Fontan Patient

open access: yesCatheterization and Cardiovascular Interventions, EarlyView.
ABSTRACT Patients with a univentricular heart following completion of the Fontan circulation often experience varying degrees of desaturation due to the formation of venovenous collaterals. We present a rare case of a Fontan patient with an enlarged azygous vein shunting into the pulmonary venous system, causing significant desaturation. As no standard
Michal Jičínský   +2 more
wiley   +1 more source

Three-Dimensional, Image-Based Evaluation of the L5 Vertebral Body and Its Ossification Center in Human Fetuses. [PDF]

open access: yesBrain Sci
Grzonkowska M   +9 more
europepmc   +1 more source

Cystatin C‐based eGFR better predicts renal vancomycin clearance than creatinine‐based eGFR in patients with allogeneic stem cell transplantation

open access: yesClinical Pharmacology &Therapeutics, EarlyView.
Knowledge of the glomerular filtration rate (GFR) is mandatory when dosing renally eliminated drugs such as vancomycin. In clinical practice, different biomarkers and various equations are used to estimate GFR (eGFR), resulting in varying estimates.
Eva‐Maria A. Wansing   +7 more
wiley   +1 more source

Lumbar region perception segmentation: a novel algorithm for automated vertebral motion parameter measurement. [PDF]

open access: yesQuant Imaging Med Surg
Zhou Z   +8 more
europepmc   +1 more source

Combinatorial expression of glial transcription factors induces Schwann cell‐specific gene expression in mouse embryonic fibroblasts

open access: yesDevelopmental Dynamics, EarlyView.
Abstract Background Schwann cells provide peripheral nerve trophic support, myelinate axons, and assist in repair. However, Schwann cell repair capacity is limited by chronic injury, disease, and aging. Schwann cell reprogramming is a cellular conversion strategy that could provide a renewable cell supply to repair injured nerves.
Lauren Belfiore   +7 more
wiley   +1 more source

Expression of mutant TIE2 p.L914F during mouse development causes embryonic lethality and defects in vascular remodeling

open access: yesDevelopmental Dynamics, EarlyView.
Abstract Background Sporadic venous malformation (VM) is associated with the hyperactivating p.L914F mutation in TIE2, a receptor tyrosine kinase essential for vascular development. This mutation is not found in hereditary VM, suggesting incompatibility with life when expressed during early vascular development.
Lindsay J. Bischoff   +6 more
wiley   +1 more source

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