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Journal of Invertebrate Pathology, 2004
Invertebrate pathologists have multiple definitions for the terms pathogenicity and virulence, and these definitions vary across disciplines that focus on host-pathogen interactions. We surveyed various literatures, including plant pathology, invertebrate pathology, evolutionary biology, and medicine, and found most define pathogenicity as the broader ...
Joseph S. Elkinton, Stephen Thomas
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Invertebrate pathologists have multiple definitions for the terms pathogenicity and virulence, and these definitions vary across disciplines that focus on host-pathogen interactions. We surveyed various literatures, including plant pathology, invertebrate pathology, evolutionary biology, and medicine, and found most define pathogenicity as the broader ...
Joseph S. Elkinton, Stephen Thomas
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Canadian Journal of Microbiology, 1973
The virulence of Nocardia brasiliensis, N. asteroides, and N. caviae was investigated. The study was done by inoculation of these organisms into the footpad of white mice. The results show that N. brasiliensis has greater virulence than N. asteroides and N. caviae. These observations resemble clinical findings in which N.
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The virulence of Nocardia brasiliensis, N. asteroides, and N. caviae was investigated. The study was done by inoculation of these organisms into the footpad of white mice. The results show that N. brasiliensis has greater virulence than N. asteroides and N. caviae. These observations resemble clinical findings in which N.
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Ecology Letters, 2006
AbstractTheoretical work has shown that parasites should evolve intermediate levels of virulence. Less attention has been given to the ecology of virulence. Here I explore population‐dynamic models of infection in an annual host. The infection does not kill the host; but it can decrease the number of offspring produced by the host, and the magnitude of
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AbstractTheoretical work has shown that parasites should evolve intermediate levels of virulence. Less attention has been given to the ecology of virulence. Here I explore population‐dynamic models of infection in an annual host. The infection does not kill the host; but it can decrease the number of offspring produced by the host, and the magnitude of
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Modifying virulent behaviour [PDF]
Cj0256-mediated phosphoethanolamine modification of lipo-oligosaccharide and the flagellar rod protein FlgG couples membrane biogenesis and motility in Campylobacter jejuni.
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2021
This thesis was scanned from the print manuscript for digital preservation and is copyright the author. Researchers can access this thesis by asking their local university, institution or public library to make a request on their behalf. Monash staff and postgraduate students can use the link in the References field.
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This thesis was scanned from the print manuscript for digital preservation and is copyright the author. Researchers can access this thesis by asking their local university, institution or public library to make a request on their behalf. Monash staff and postgraduate students can use the link in the References field.
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Trends in Microbiology, 1994
Why is there variation in the virulence of infectious diseases? Virulence can have substantial effects on the genetic contribution of both host and pathogen to future generations. Understanding it therefore requires explanation not only in terms of cellular and molecular mechanisms, but also in evolutionary terms: what is the nature of the selection ...
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Why is there variation in the virulence of infectious diseases? Virulence can have substantial effects on the genetic contribution of both host and pathogen to future generations. Understanding it therefore requires explanation not only in terms of cellular and molecular mechanisms, but also in evolutionary terms: what is the nature of the selection ...
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Trends in Microbiology, 2002
Why do viruses cause disease? As intracellular parasites they grow at the expense of the host, yet many infections are non-virulent. We tend to focus on unusual outcomes of infection that are important to the individual but trivial for host-parasite evolution, for example, paralytic polio or viral cancer.
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Why do viruses cause disease? As intracellular parasites they grow at the expense of the host, yet many infections are non-virulent. We tend to focus on unusual outcomes of infection that are important to the individual but trivial for host-parasite evolution, for example, paralytic polio or viral cancer.
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Nursing Standard, 1988
The virulence of human immunodeficiency virus may increase in the later stages of the disease, which might explain why people go on to get full blown AIDS.
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The virulence of human immunodeficiency virus may increase in the later stages of the disease, which might explain why people go on to get full blown AIDS.
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