Results 141 to 150 of about 60,298 (290)
JDDG: Journal der Deutschen Dermatologischen Gesellschaft, EarlyView.
Charbel Skayem +7 more
wiley +1 more source
Interactome analysis of gene expression profile reveals potential novel key transcriptional regulators of skin pathology in vitiligo [PDF]
Rama Dey-Rao, Animesh A. Sinha
openalex +1 more source
JDDG: Journal der Deutschen Dermatologischen Gesellschaft, EarlyView.
Yonit Wohl +4 more
wiley +1 more source
ABSTRACT The etiology of ankylosing spondylitis (AS) is complex and not yet fully understood. Interleukin‐6 (IL‐6), vitamin D and the vitamin D receptor (VDR) play an important role in modulating immune response, and Torque teno virus is considered a marker of immune status. This case–control study aimed to investigate the predisposition to AS. A total
Dănuț Cimponeriu +6 more
wiley +1 more source
609P The association between thyroid function abnormalities and vitiligo induced by pembrolizumab regarding prognosis in patients with advanced melanoma [PDF]
Moez Jawad Mobarek +3 more
openalex +1 more source
Tissue Resident Memory Cells: Friend or Foe?
Tissue‐resident memory T cells (TRM cells) are specialised immune cells in barrier tissues like the lungs, skin and gut, providing rapid host defence and tumour surveillance. Their retention and differentiation are regulated by molecules such as CD69, CD103 and TGF‐β. Dysregulation of TRM cells can lead to chronic activation, driving conditions such as
Chidimma F. Chude +2 more
wiley +1 more source
Physician burnout or joy: Rediscovering the rewards of a life in medicine,
Pearl E. Grimes, MD
doaj +1 more source
Treatment advances in Vitiligo: An Updated Review
Introduction Vitiligo is a common disorder of depigmentation caused by the progressive destruction of melanocytes that affects the skin, hair, and mucous membranes, clinically presenting as depigmented macules and leukotrichia.
Ishrat Binti Ismail +2 more
doaj +1 more source
A constant rate of DNA damage that is not perfectly repaired will cause a constant rate of DNA mutations. The chance of mutation will increase if DNA is prone to damage, such as occurs in somatic hypermutation (SHM) hotspots and GC‐rich DNA. Thus, if one mutation‐prone DNA site drives disease, the age of onset of disease and degree of penetrance should
Piet C. de Groen
wiley +1 more source

