Results 121 to 130 of about 9,619 (201)
African horsesickness virus serotype 4 (AHSV-4) outer capsid protein VP2, or VP2 and VP5 plus inner capsid protein VP7, derived from single or dual recombinant baculovirus expression vectors were used in different combinations to immunize horses.
Martínez-Torrecuadrada, Jorge Luis +6 more
core +1 more source
(A) Schematic illustration of an infectious rotavirus particle. Subunits are shown in surface representation and the particle is partially cut to allow inside view.
Stephen C. Harrison (9731984) +4 more
core +1 more source
Classification #3 and #4 of reversed VP5*/VP8* spikes.
Classification #3 and #4 of reversed VP5*/VP8* spikes.
Stephen C. Harrison (9731984) +4 more
core +1 more source
Bluetongue virus (BTV) is transmitted by blood-feeding insects (Culicoides sp.) and causes hemorrhagic diseases in livestock. BTV is a nonenveloped, double-stranded RNA (dsRNA) virus with two capsids: a well-studied, stable core enclosing the dsRNA ...
Boyce, Mark +6 more
core +1 more source
Expression Studies of VP2 and VP5 Outer Capsid Proteins of Bluetongue Virus
Resumen en inglès, ver archivo ...
F. Mohd Jaafar +4 more
openaire +1 more source
Epitopic Mapping of Serogroup and Serotype Specific Proteins, VP7 and VP5, Among Five U.S. Bluetongue Viruses [PDF]
The identification of antigenic epitopes on VP7 and VP5, serogroup and serotype-specific antigens of bluetongue virus (BTV), respectively, was studied using the combination of polyclonal, monoclonal and oligoclonal antibodies and competitive blocking ...
Yang, Yi-Yuan
core
Plasmid DNA-based reverse genetics as a platform for manufacturing of bluetongue vaccine
Control of bluetongue disease is predominantly through vaccination with licensed inactivated or live-attenuated vaccines (LAVs). Manufacturing of LAVs in endemic countries requires formulation with a high number of serotypes for effective protection ...
Tendai A. M. Mlingo +2 more
doaj +1 more source
Effect of VP5 abrogation on the IBDV replication program.
QM7 cell monolayers were mock-infected (Mock) or infected (MOI of 3 PFU/cell) with the WT or the VP5-KO virus. (A) Accumulation kinetics of IBDV-specific RNAs. Cultures harvested at the indicated times PI were used for the isolation of total RNA.
Fernando Méndez (725779) +5 more
core +1 more source
Analysis of function and immunogenicity of GCRV-GD108 VP5
Hangjun WANG +4 more
openaire +1 more source
VP5 C-terminal ablation affects the IBDV plaque size phenotype.
A. Plaque assays. QM7 cell monolayers were infected with the VP5 knockout mutant (KO) virus, the wild type (WT) virus and mutant IBDV viruses lacking 3 (Δ3CT), 10 (Δ10CT), and 14 (Δ14CT) VP5 C-terminal residues, respectively.
Fernando Méndez (725779) +3 more
core +1 more source

