Results 41 to 50 of about 60,487 (208)
Treatment Outcomes of Ocular Manifestations in Wernicke\u27s Encephalopathy: Case Report
The case of a 28- year-old woman with a past medical history of gastric sleeve who was reported to have blurry vision and presented to neuro-ophthalmology with double vision. On examination the patient had bilateral abducens palsy, alternating upbeat and
Whitney Stuard Sambhariya, PhD, Medical Student; Melanie Truong-Le, DO, OD
core
Abstract Introduction Fluid restriction is the current first‐line therapy for hyponatremia induced by the syndrome of inappropriate antidiuresis (SIAD), yet up to 50% of patients do not achieve an adequate plasma sodium correction. Reported predictors of nonresponse include urine osmolality >500 mOsm/kg, the Furst ratio >1 [(UNa + UK)/(PNa)], urine ...
Michelle Patricia Müller +5 more
wiley +1 more source
About pathognomonic images: an infrequent case of acute encephalopathy
BACKGROUND The occurrence of acute encephalopathy is a dramatic clinical dilemma when usual diagnostic techniques (blood tests, cerebral CT and cerebrospinal fluid analysis) show no abnormalities.
Alessandro Grasso +3 more
doaj +1 more source
Wernicke encephalopathy: An updated narrative review
Wernicke's encephalopathy (WE) and Korsakoff Syndrome (KS) are distinct neurological disorders that may have overlapping clinical features. Due to the overlap, they are collectively known as Wernicke–Korsakoff syndrome.
Elmukhtar Habas +4 more
doaj +1 more source
ABSTRACT Bariatric surgery is increasingly performed in adolescents and young adults. Although sleeve gastrectomy preserves intestinal continuity and is often regarded as nutritionally low‐risk, it can still precipitate micronutrient deficiency, and obesity itself predisposes to thiamine deficiency, which can cause both Wernicke encephalopathy and a ...
Altin Kuqo, Floreta Kurti, Erand Llanaj
wiley +1 more source
Wernicke’s encephalopathy and Korsakoff’s psychosis are the acute and chronic phases of Wernicke–Korsakov syndrome, a neurodegenerative process that is caused by thiamine deficiency and predominantly develops in persons with severe alcohol problems.
Yu. P. Sivolap, I. V. Damulin
doaj +1 more source
The fortification of white maize flour with jackfruit seed powder significantly enhanced the nutritional value of stiff maize porridge. The increase in the proportion of jackfruit seed powder in maize‐jackfruit blended flours resulted in increased nutritional quality in the corresponding stiff maize porridge.
Felix Ochieng Odhoch +6 more
wiley +1 more source
Gayet-Wernicke encephalopathy (GWE) is a neuropsychiatric syndrome due to Vitamin B1 (thiamine) deficiency, fatal in 30% of cases and preventable if treatment is initiated early, characterized by the classic triad of encephalopathy, ocular involvement ...
Hicham Ziani +6 more
doaj +1 more source
Effect of GLP‐1 receptor agonist on nutrient intake: A narrative review
Abstract Humans have a primitive defense mechanism called metabolic adaptation that slows voluntary weight loss. It works by downregulating hormones that reduce food intake and by lowering the metabolic rate. Second‐generation GLP‐1 receptor agonists, such as semaglutide or tirzepatide, promote weight loss by targeting this hormonal system to control ...
Ken Fujioka
wiley +1 more source
Wernicke encephalopathy: a literature review. [PDF]
Background. Wernicke encephalopathy (WE) is an acute neurological syndrome caused by thiamine deficiency. First described by Karl Wernicke in 1881, the disease is characterized by a triad of symptoms—encephalopathy, ataxia, and ocular disturbances. WE is
Nasvytis, Matas, +1 more
core +1 more source

