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内质网应激通过抑制PI3K/AKT/mTOR信号通路诱导多形性胶质母细胞瘤干细胞凋亡的机制

open access: yesZhongshan Daxue xuebao. Yixue kexue ban, 2016
摘 要: 【目的】 探索内质网应激(ER stress)通过抑制PI3K/AKT/mTOR信号通路诱导多形性胶质母细胞瘤肿瘤干细胞(GSC)凋亡的机制?【方法】 临床获取多形性胶质母细胞瘤肿瘤组织标本,剪碎?胰酶消化?过滤?去除红细胞后,培养GSC;免疫荧光技术检测GSC抗原CD133?GFAP? Nestin?
doaj  

[Indolent γδT-cell clone in Felty syndrome: a case report and literature review]. [PDF]

open access: yesZhonghua Xue Ye Xue Za Zhi
Wang C, Fu MW, An G, Huang WY, Yang LJ.
europepmc   +1 more source

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