High frequency deep brain stimulation of the hippocampus in a rat model for temporal lobe epilepsy [PDF]
Boon, Paul +9 more
core +1 more source
Objective To assess real‐world effectiveness of switching disease‐modifying therapy (DMT) in pediatric multiple sclerosis (MS) and clinically isolated syndrome (CIS) initially treated with platform injectables on disease activity. Methods Of 2615 pediatric‐onset demyelinating disease patients at 12 clinics in the United States (US) Network of Pediatric
Aaron W. Abrams +27 more
wiley +1 more source
Striatum is the potential target for treating absence epilepsy: a theoretical evidence. [PDF]
Hu B, Zhou W, Ma X.
europepmc +1 more source
EEG Oscillatory Networks in Peri-Ictal Period of Absence Epilepsy. [PDF]
Li Z +6 more
europepmc +1 more source
Childhood absence epilepsy: Behavioral, cognitive, and linguistic comorbidities [PDF]
Rochelle Caplan +8 more
openalex +1 more source
Absence epilepsy: Continuum of clinical presentation and epigenetics?
L. Guilhoto
semanticscholar +1 more source
Harnessing the Neurobiology of Empathy and Compassion to Alleviate Burnout in Neurology
The neurologist cares for patients with complex chronic conditions that compromise their wellbeing. Many such disorders are poorly understood. Whereas for some, effective symptomatic treatments exist, for most we lack an understanding of the molecular and cellular bases for disease pathogenesis needed for discovering disease‐modifying treatments ...
Fadel Zeidan +2 more
wiley +1 more source
Attentional Deficits and Absence Epilepsy: A Tale of 2 Interneuronopathies. [PDF]
Knowles J.
europepmc +1 more source
The Orexin System: A Potential Player in the Pathophysiology of Absence Epilepsy. [PDF]
Celli R, Luijtelaar GV.
europepmc +1 more source
Anatomical Progression of Neuropathology in FTLD‐TDP Type C and Linkage to Annexin A11
Objective Frontotemporal lobar degenerations (FTLD)‐TDP type C (TDP‐C) is distinguished from other FTLD‐TDP subtypes by 3 unique features: (1) invariable onset in the anterior temporal lobe (ATL), (2) phosphorylated TDP‐43 (pTDP) neurites in cortex, and (3) colocalization of all pTDP deposits with annexin A11 (ANXA11).
Allegra Kawles +7 more
wiley +1 more source

