Results 271 to 280 of about 2,037,182 (380)
Suppression of seizure in childhood absence epilepsy using robust control of deep brain stimulation: a simulation study. [PDF]
Rouhani E, Jafari E, Akhavan A.
europepmc +1 more source
SCN3B encodes the β3 auxiliary subunit, essential for voltage‐gated Na+ (Nav) channel trafficking and gating. Although SCN3B has been associated with cardiac disorders, a link with neurodevelopmental disorders (NDD) has not been established. Using a genotype‐first approach, we identified homozygous truncating variants (c.281G>A‐β3W94*, c.584 + 1G>A ...
Nathan Routledge +11 more
wiley +1 more source
A 6-year-old with childhood absence epilepsy and motor hyperactivity. [PDF]
Auvin S.
europepmc +1 more source
Erratum: Age-dependent decline in learning and memory performances of WAG/Rij rat model of absence epilepsy [PDF]
Ayşe Karson +4 more
openalex +1 more source
High frequency deep brain stimulation of the hippocampus in a rat model for temporal lobe epilepsy [PDF]
Boon, Paul +9 more
core +1 more source
Objective In Parkinson's disease (PD), the power of beta oscillations (± 13–30 hertz [Hz]) from subthalamic nucleus (STN) local field potentials (LFPs) is associated with motor symptoms. Beta power can be used for adaptive deep brain stimulation (aDBS) algorithms based upon symptom fluctuations.
Martijn G.J. de Neeling +8 more
wiley +1 more source
Structural Abnormalities in Childhood Absence Epilepsy: Voxel-Based Analysis Using Diffusion Tensor Imaging [PDF]
Wenchao Qiu +8 more
openalex +1 more source
Absence epilepsy: Continuum of clinical presentation and epigenetics?
L. Guilhoto
semanticscholar +1 more source
Objective Impaired ability to induce stepping after incomplete spinal cord injury (SCI) can limit the efficacy of locomotor training, often leaving patients wheelchair‐bound. The cuneiform nucleus (CNF), a key mesencephalic locomotor control center, modulates the activity of spinal locomotor centers via the reticulospinal tract.
Anna‐Sophie Hofer +21 more
wiley +1 more source

