Results 41 to 50 of about 758,569 (257)

Evaluation Method for Highly Cited Scholars based on CNKI data

open access: yesZhishi guanli luntan
[Purpose/Significance] This study aims to measure the influence of scholars objectively and scientifically, identify a group of scholars with rich scientific research achievements and outstanding academic influence in China in the past 10 years, and ...
Wu Junhong, Zheng Xinyu, Sun Jun, Xiao Hong, Tang Liyun
doaj   +1 more source

Open government partnership as a platform for advancing open education policy

open access: yesOpen Praxis, 2015
An exciting new avenue for establishing and expanding national commitments to open education has emerged through the Open Government Partnership (OGP), a multilateral initiative that aims to secure commitments from governments to make their governance ...
Jan Gondol, Nicole Allen
doaj   +3 more sources

The Future of Pediatric Hematology‐Oncology Fellowship Training: Reflections on the American Board of Pediatrics Training Model

open access: yes
Pediatric Blood &Cancer, EarlyView.
Scott C. Borinstein   +11 more
wiley   +1 more source

Transferrin receptor 1‐mediated iron uptake supports thermogenic activation in human cervical‐derived adipocytes

open access: yesFEBS Letters, EarlyView.
In this study, we found that human cervical‐derived adipocytes maintain intracellular iron level by regulating the expression of iron transport‐related proteins during adrenergic stimulation. Melanotransferrin is predicted to interact with transferrin receptor 1 based on in silico analysis.
Rahaf Alrifai   +9 more
wiley   +1 more source

The Enclosure and Alienation of Academic Publishing: Lessons for the Professoriate

open access: yestripleC: Communication, Capitalism & Critique, 2012
This paper interrogates and situates theoretically from a Marxist perspective various aspects and tensions that inhere in the contemporary academic publishing environment. The focus of the article is on journal publishing.
Wilhelm Peekhaus
doaj   +1 more source

The Scandal of Academic Publishing

open access: yesPublishing Research Quarterly
Abstract Much is rotten in academic publishing, but it is easier to hold noses than do anything more fundamental about the stench. The five companies dominating the industry have grown fat on the backs of free academic labour. Why do academics (and so their research funders, their employers and the taxpayer) continue to subsidise the ...
openaire   +2 more sources

Peripheral lysosomes recruit PLEKHG3 to focal adhesions and restrain protrusion dynamics

open access: yesFEBS Letters, EarlyView.
Proximity‐dependent labeling at the LAMTOR complex revealed the Rho GEF PLEKHG3 as a lysosome‐proximal protein directing the study toward the influence of lysosome positioning on actin dynamics and cell motility. We show that PLEKHG3 colocalizes with lysosomes at focal adhesion sites and observe that forced peripheral dispersion of lysosomes hinders ...
Rainer Ettelt   +8 more
wiley   +1 more source

Finding novel vulnerabilities of hypomorphic BRCA1 alleles

open access: yesMolecular Oncology, EarlyView.
Synthetic lethality screens performed to identify novel vulnerabilities often model complete gene loss, thereby overlooking patient‐derived hypomorphic mutations. In this study, we have performed genome‐wide CRISPR screens on BRCA1 hypomorphic mutations, showing BRCA1I26A behaves like wild‐type, while BRCA1R1699Q mimics deficiency. Furthermore, we have
Anne Schreuder   +10 more
wiley   +1 more source

Predators and hijackers in academic publishing

open access: yesEuropean Journal of General Practice, 2015
A theme that has recently received increasing attention in medical publishing is predatory journals.
Igor, Švab, Irena, Makivić
openaire   +2 more sources

Oncogenic DMTF1β promotes cancer cell motility by regulating autophagy through ULK1 stabilization

open access: yesMolecular Oncology, EarlyView.
In the current study, we demonstrate that the oncogene DMTF1β regulates ULK1 stability by reducing its proteasomal degradation in cancer cells. This stabilization enables ULK1 to induce autophagy, which in turn facilitates cancer cell migration. Consequently, reduced DMTF1β levels lead to decreased autophagy and impaired cancer cell migration.
Jun Xu   +13 more
wiley   +1 more source

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