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Pseudo-Renal Tubular Acidosis: Conditions Mimicking Renal Tubular Acidosis
Advances in Chronic Kidney Disease, 2018 Hyperchloremic metabolic acidosis, particularly renal tubular acidosis, can pose diagnostic challenges. The laboratory phenotype of a low total carbon dioxide content, normal anion gap, and hyperchloremia may be misconstrued as hypobicarbonatemia from renal tubular acidosis.
Jerry Yee
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Kidney International, 2020 Distal renal tubular acidosis is a rare renal tubular disorder characterized by hyperchloremic metabolic acidosis and impaired urinary acidification. Mutations in three genes (ATP6V0A4, ATP6V1B1 and SLC4A1) constitute a monogenic causation in 58-70% of ...
Tilman Jobst-Schwan +2 more
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Renal tubular acidosis: developments in our understanding of the molecular basis
International Journal of Biochemistry and Cell Biology, 2005 Renal tubular acidosis is a metabolic acidosis due to impaired acid excretion by the kidney. Hyperchloraemic acidosis with a normal anion gap and normal (or near normal) glomerular filtration rate, and in the absence of diarrhoea, defines this disorder ...
Ashley M Toye +2 more
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Pediatrics In Review, 2017
1. Jonathan Pelletier, MD* 2. Rasheed Gbadegesin, MD, MBBS† 3. Betty Staples, MD* 1. *Department of Pediatrics and 2. †Department of Pediatric Nephrology, Duke Children's Hospital and Health Center, Durham, NC 1. 1. Reddy P Clinical Approach to Renal Tubular Acidosis in Adult Patients. Reddy P Int J Clin Pract. 2011;65(3):350–360
Jonathan, Pelletier +2 more
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1. Jonathan Pelletier, MD* 2. Rasheed Gbadegesin, MD, MBBS† 3. Betty Staples, MD* 1. *Department of Pediatrics and 2. †Department of Pediatric Nephrology, Duke Children's Hospital and Health Center, Durham, NC 1. 1. Reddy P Clinical Approach to Renal Tubular Acidosis in Adult Patients. Reddy P Int J Clin Pract. 2011;65(3):350–360
Jonathan, Pelletier +2 more
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The Indian Journal of Pediatrics, 2015
Renal tubular acidosis (RTA) comprises a group of disorders characterized by low capacity for net acid excretion and persistent hyperchloremic metabolic acidosis, despite preserved glomerular filtration rate. RTA are classified into chiefly three types (1, 2 and 4) based on pathophysiology and clinical and laboratory characteristics. Most patients have
Arvind Bagga, Aditi Sinha
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Renal tubular acidosis (RTA) comprises a group of disorders characterized by low capacity for net acid excretion and persistent hyperchloremic metabolic acidosis, despite preserved glomerular filtration rate. RTA are classified into chiefly three types (1, 2 and 4) based on pathophysiology and clinical and laboratory characteristics. Most patients have
Arvind Bagga, Aditi Sinha
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Pediatric Nephrology, 1990
The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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Journal of Nephrology, 2006
Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Journal of Urology, 1984
Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Medical Clinics of North America, 1983
The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1999
Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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