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Survival of Actinobacillus pleuropneumoniae outside the pig

Research in Veterinary Science, 2013
Transmission of Actinobacillus pleuropneumoniae is primarily thought to be via direct transfer of mucus from pig to pig. For transfer between farms, the organism may need to persist in the wet or dried state to be carried on an inanimate surface. The survival of A. pleuropneumoniae was investigated under controlled laboratory conditions.
P, Assavacheep, A N, Rycroft
openaire   +2 more sources

Actinobacillus pleuropneumoniae: pathobiology and pathogenesis of infection

Microbes and Infection, 2002
Actinobacillus pleuropneumoniae causes porcine pleuropneumonia, a highly contagious disease for which there is no effective vaccine. This review considers how adhesins, iron-acquisition factors, capsule and lipopolysaccharide, RTX cytotoxins and other potential future vaccine components contribute to colonisation, to avoidance of host clearance ...
Janine T, Bossé   +6 more
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Hemagglutinating properties of Actinobacillus pleuropneumoniae

Canadian Journal of Microbiology, 1988
A total of 26 isolates of Actinobacillus pleuropneumoniae were tested for their ability to agglutinate erythrocytes of different origins. Seven different hemagglutination patterns were found. Ten (38%) isolates did not agglutinate any of the erythrocytes tested.
M, Jacques, G, Roy, K R, Mittal
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A targeted mutagenesis system for Actinobacillus pleuropneumoniae

Gene, 1995
We describe methods for the mutagenesis of cloned Actinobacillus pleuropneumoniae (Ap) genes and for the construction of Ap mutants by allelic exchange. We used these methods to construct isogenic mutants of Ap which no longer synthesized a 48-kDa outer membrane protein (AopA).
M H, Mulks, J M, Buysse
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Screening of Actinobacillus pleuropneumoniae LuxS Inhibitors

Current Microbiology, 2013
LuxS, a conserved bacterial enzyme involved in the activated methyl cycle, catalyzes S-ribosylhomocysteine (SRH) into homocysteine and AI-2 (the inter-species quorum-sensing signal molecule). This enzyme has been reported to be essential for the survival of Actinobacillus pleuropneumoniae in its natural host.
Lu, Li   +6 more
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Design and analysis of an Actinobacillus pleuropneumoniae transmission experiment

Preventive Veterinary Medicine, 2003
This paper describes a methodology to quantify the transmission of Actinobacillus (A.) pleuropneumoniae from subclinically infected carrier pigs to susceptible contact pigs, and to test the effect of possible interventions on the transmission. The methodology includes the design of a transmission experiment, and a method with which A.
Velthuis, A.G.J.   +4 more
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Effects of fisetin on virulence of Actinobacillus Pleuropneumoniae

Microbial Pathogenesis
Porcine infectious pleuropneumonia (PCP), caused by Actinobacillus pleuropneumoniae (APP), is a highly infectious respiratory disease of pigs, resulting in significant economic losses to the industry. Apx toxins are cytolytic virulence factors produced by APP.
Qiyun He   +7 more
openaire   +2 more sources

Exotoxins of Actinobacillus Pleuropneumoniae

1995
Actinobacillus pleuropneumoniae (previously Haemophilus pleuropneumoniae) is the etiological agent of swine pleuropneumonia, a severe, contagious disease of swine causing important economic losses in industrialized swine production worldwide. The disease is either acute and often fatal or chronic localized and necrotizing.
openaire   +1 more source

Virulence in Actinobacillus pleuropneumoniae and RTX toxins

Trends in Microbiology, 1995
RTX toxins are pore-forming, cytolytic protein toxins that occur widely among pathogenic Gram-negative bacteria. RTX toxins appear to play a direct role in the virulence of Actinobacillus pleuropneumoniae, the etiological agent of porcine pleuropneumonia.
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Characterization and immunogenicity of an apxIA mutant of Actinobacillus pleuropneumoniae

Veterinary Microbiology, 2006
Actinobacillus pleuropneumoniae is the aetiological agent of porcine pleuropneumonia, a highly contagious and often fatal disease. A candidate live vaccine strain, potentially capable of cross-serovar protection, was constructed by deleting the section of the apxIA gene coding for the C-terminal segment of ApxI toxin of the A.
Xu, Fuzhou   +8 more
openaire   +4 more sources

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