Results 111 to 120 of about 3,639,746 (361)

PARALYSES FOLLOWING ACUTE DISEASES. [PDF]

open access: yesJAMA: The Journal of the American Medical Association, 1892
There are some varieties of paralyses that follow acute diseases which present some features of great interest. This interest can never cease until the true pathology underlying these cases has been fully worked out. My remarks on the present occasion are with the view, that though they may not settle, they may suggest the lines on which some of these ...
openaire   +3 more sources

Comparing self‐reported race and genetic ancestry for identifying potential differentially methylated sites in endometrial cancer: insights from African ancestry proportions using machine learning models

open access: yesMolecular Oncology, EarlyView.
Integrating ancestry, differential methylation analysis, and machine learning, we identified robust epigenetic signature genes (ESGs) and Core‐ESGs in Black and White women with endometrial cancer. Core‐ESGs (namely APOBEC1 and PLEKHG5) methylation levels were significantly associated with survival, with tumors from high African ancestry (THA) showing ...
Huma Asif, J. Julie Kim
wiley   +1 more source

EPI Update: Special Alert, July 3, 2013 [PDF]

open access: yes, 2013
Weekly newsletter for Center For Acute Disease Epidemiology of Iowa Department of Public Health.

core  

Disease severity adversely affects delivery of dialysis in acute renal failure [PDF]

open access: yes, 2007
Background/Aims: Methods of intermittent hemodialysis (IHD) dose quantification in acute renal failure (ARF) are not well defined. This observational study was designed to evaluate the impact of disease activity on delivered single pool Kt/V-urea in ARF ...
American College of Chest Physicians/Society of Critical Care Medicine Consensus Conference   +20 more
core   +1 more source

IS ACUTE CHOREA AN INFECTIOUS DISEASE? [PDF]

open access: yesJAMA: The Journal of the American Medical Association, 1910
The frequent (though not constant) association of chorea with acute inflammatory rheumatism is a well-established fact. Whether the infectious element of the latter ( Micrococcus rheumaticus , streptococci) or the toxins elaborated by the suspected micro-organism are the same etiologic causes of the acute chorea it is impossible as yet to ascertain ...
openaire   +2 more sources

Multidimensional OMICs reveal ARID1A orchestrated control of DNA damage, splicing, and cell cycle in normal‐like and malignant urothelial cells

open access: yesMolecular Oncology, EarlyView.
Loss of the frequently mutated chromatin remodeler ARID1A, a subunit of the SWI/SNF cBAF complex, results in less open chromatin, alternative splicing, and the failure to stop cells from progressing through the cell cycle after DNA damage in bladder (cancer) cells. Created in BioRender. Epigenetic regulators, such as the SWI/SNF complex, with important
Rebecca M. Schlösser   +11 more
wiley   +1 more source

Psychological status and recurrent aphthous ulceration [Psihološki status i rekurentne aftozne ulceracije] [PDF]

open access: yes, 2012
Recurrent aphthous ulceration (RAU) are a disease of an unknown etiology and mediated through T-cell lymphocytes. Evidence suggests that RAU is connected with chronic bowel disease, haematinic deficiencies, AIDS, food hypersensitivity and severe stress ...
Andabak Rogulj, Ana   +7 more
core  

Coexisting Sickle Cell Anemia and Sarcoidosis: A Management Conundrum! [PDF]

open access: yes, 2017
Sickle cell disease and Sarcoidosis are conditions that are more common in the African American population. In this report we share an unfortunate patient who had hepatic sarcoidosis but could not receive steroids since that precipitated acute liver ...
Gollahalli, Nagesh S., Nutan, FNU
core   +3 more sources

Remissions in leukemia of childhood following acute infectious disease. Staphylococcus and streptococcus, varicella, and feline panleukopenias [PDF]

open access: bronze, 1953
Howard R. Bierman   +6 more
openalex   +1 more source

Escape from TGF‐β‐induced senescence promotes aggressive hallmarks in epithelial hepatocellular carcinoma cells

open access: yesMolecular Oncology, EarlyView.
Chronic TGF‐β exposure drives epithelial HCC cells from a senescent state to a TGF‐β resistant mesenchymal phenotype. This transition is characterized by the loss of Smad3‐mediated signaling, escape from senescence, enhanced invasiveness and metastatic potential, and upregulation of key resistance modulators such as MARK1 and GRM8, ultimately promoting
Minenur Kalyoncu   +11 more
wiley   +1 more source

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