Results 41 to 50 of about 24,673 (247)

Temporary cardiac pacing induced electrocardiographic changes simulating myocardial infarction [PDF]

open access: yes, 2017
Temporary transvenous pacing is an immediate lifesaving measure in patients with Stokes-Adams syndrome and in patients with symptomatic bradycardia. Bradyarrhythmias are known to occur in acute myocardial infarction. But in a paced heart, it is difficult
Behera, Suresh Kumar   +2 more
core   +2 more sources

Association of Race and Ethnicity With Medication Use for Pediatric Lupus in the Childhood Arthritis and Rheumatology Research Alliance Registry

open access: yesACR Open Rheumatology, Volume 4, Issue 11, Page 954-963, November 2022., 2022
Objective Black and Hispanic children with pediatric lupus (pSLE) have higher morbidity and mortality than non‐Hispanic White children. The extent to which differences in outcomes are due to treatment disparities, including medication use, is unknown.
Jordan E. Roberts   +4 more
wiley   +1 more source

The DNA-binding domain of the Chd1 chromatin-remodelling enzyme contains SANT and SLIDE domains [PDF]

open access: yes, 2011
The ATP-dependent chromatin-remodelling enzyme Chd1 is a 168-kDa protein consisting of a double chromodomain, Snf2-related ATPase domain, and a C-terminal DNA-binding domain.
Aasland   +65 more
core   +3 more sources

Ventricular standstill disguised as epilepsy: A case report on Stokes-Adams attacks

open access: yesHeliyon, 2023
Background: Stokes-Adams attacks presenting as convulsions may be difficult to distinguish from epilepsy. Stokes-Adams Syndrome is a transient abrupt collapse into unconsciousness due to a sudden but pronounced decrease in cardiac output caused by change
Mykha Marie B. Tabuzo   +2 more
doaj  

Gain of function mutants: Ion channels and G protein-coupled receptors [PDF]

open access: yes, 2000
Many ion channels and receptors display striking phenotypes for gain-of-function mutations but milder phenotypes for null mutations. Gain of molecular function can have several mechanistic bases: selectivity changes, gating changes including constitutive
Karschin, Andreas, Lester, Henry A.
core   +1 more source

Heart transplantation in a patient with primary malignant tumor

open access: yesВестник трансплантологии и искусственных органов, 2019
The article looks at a clinical case of a 31-year-old female with signs of paroxysmal ventricular tachycardia and Morgagni–Adams–Stokes syndrome. In April 2014, electrophysiological examination revealed a source of ventricular tachycardia localized in ...
E. E. Kliver   +4 more
doaj   +1 more source

Syndrome decoding by quantum approximate optimization [PDF]

open access: yesQuantum Inf Process 23, 368 (2024), 2022
The syndrome decoding problem is known to be NP-complete. The goal of the decoder is to find an error of low weight that corresponds to a given syndrome obtained from a parity-check matrix. We use the quantum approximate optimization algorithm (QAOA) to address the syndrome decoding problem with elegantly-designed reward Hamiltonians based on both ...
arxiv   +1 more source

Adaptive syndrome measurements for Shor-style error correction [PDF]

open access: yesQuantum 7, 1075 (2023), 2022
The Shor fault-tolerant error correction (FTEC) scheme uses transversal gates and ancilla qubits prepared in the cat state in syndrome extraction circuits to prevent propagation of errors caused by gate faults. For a stabilizer code of distance $d$ that can correct up to $t=\lfloor(d-1)/2\rfloor$ errors, the traditional Shor scheme handles ancilla ...
arxiv   +1 more source

Quantum Data-Syndrome Codes [PDF]

open access: yesIEEE Journal on Selected Areas in Communications, vol. 38, no. 3, pp. 449-462, March 2020, 2019
Performing active quantum error correction to protect fragile quantum states highly depends on the correctness of error information--error syndromes. To obtain reliable error syndromes using imperfect physical circuits, we propose the idea of quantum data-syndrome (DS) codes that are capable of correcting both data qubits and syndrome bits errors.
arxiv   +1 more source

RPGR protein complex regulates proteasome activity and mediates store-operated calcium entry [PDF]

open access: yes, 2018
Ciliopathies are a group of genetically heterogeneous disorders, characterized by defects in cilia genesis or maintenance. Mutations in the RPGR gene and its interacting partners, RPGRIP1 and RPGRIP1L, cause ciliopathies, but the function of their ...
Aguirre   +68 more
core   +3 more sources

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