Results 81 to 90 of about 31,072 (141)

Efficacy of Baricitinib in Moderate Alopecia Areata, Severe Alopecia Areata, Alopecia Totalis, and Alopecia Universalis: A Real-world Comparative Study.

open access: yes
This dataset supports the findings in the manuscript "Efficacy of Baricitinib in Moderate Alopecia Areata, Severe Alopecia Areata, Alopecia Totalis, and Alopecia Universalis: A Real-world Comparative Study." The dataset includes:Demographic and baseline ...
Jiang, Guan
core   +1 more source

Three mutations in SASH1 cause the pathogenesis of dyschromatosis universalis hereditaria (DUH) [PDF]

open access: yes, 2011
Dyschromatosis universalis hereditaria (DUH) is a rare genodermatosis characterized by hyper- and hypopigmented macules which form a reticulate or mottled pattern.
Zhiyun Wei, Qinghe Xing, Dingan Zhou
core  

Alopecia areata universalis showing excellent response to combination treatment modalities

open access: yes, 2022
Alopecia areata (AA) is a non-scarring alopecia with mainly an autoimmune etiopathogenesis. The most severe AA variant is alopecia areata universalis (AAU), characterized by a total loss of scalp and body hair. AAU poses a significant psychosocial impact
Noer Kamila   +4 more
core   +1 more source

A mouse model of clonal CD8+ T lymphocyte-mediated alopecia areata progressing to alopecia universalis.

open access: yes, 2012
Alopecia areata is among the most prevalent autoimmune diseases, yet compared with other autoimmune conditions, it is not well studied. This in part results from limitations in the C3H/HeJ mouse and DEBR rat model systems most commonly used to study the ...
Alli, Rajshekhar   +4 more
core   +3 more sources

Complete remission of alopecia universalis after allogeneic hematopoietic stem cell transplantation.

open access: yes, 2005
This case report is on a 40-year-old male patient with chronic myeloid leukemia (CML) receiving an allogeneic hematopoietic stem cell transplantation (HSCT) in first chronic phase from an HLA-identical sibling brother.
Gratwohl A   +7 more
core   +1 more source

Ritlecitinib: Efficacy of a Novel Therapy for Severe Alopecia Areata in Patients Aged 12 Years and Older

open access: yesActas Dermo-Sifiliográficas
Alopecia areata (AA) is a dermatological disease of immune origin characterized by partial or total hair loss of the scalp (alopecia totalis) or the whole body (alopecia universalis).
S. Vañó-Galván   +5 more
doaj   +1 more source

Molecular signatures define alopecia areata subtypes and transcriptional biomarkers

open access: yesEBioMedicine, 2016
Alopecia areata (AA) is an autoimmune disease typified by nonscarring hair loss with a variable clinical course. In this study, we conducted whole genome gene expression analysis of 96 human scalp skin biopsy specimens from AA or normal control subjects.
Ali Jabbari   +9 more
doaj   +1 more source

Of Hairless Mice and Men: The Genetic Basis of Congenital Alopecia Universalis/Congenital Atrichia

open access: yes, 1999
Background: Mouse models of human diseases help identify gene defects. Objective: The methods of homozygosity mapping and mouse/human homology to identify genes are reviewed.
Sherri J. Bale
core   +1 more source

Association between alopecia areata and cardiovascular disease: a systematic review and meta-analysis

open access: yesFrontiers in Immunology
BackgroundAlopecia areata (AA) is a common autoimmune disorder causing patchy hair loss. Epidemiological observations and molecular studies collectively suggest an underrecognized interplay between AA and cardiovascular disease (CVD).
Jiawei Lu   +4 more
doaj   +1 more source

Alopecia Universalis Causes Serious Personality Derangements: First Report of Negative Impact of a Medical Condition on Personality (279 Patients)

open access: yes, 2011
ObjectiveTo evaluate the reason of major behavioral problems in alopecia universalis patients, we tried to examine by way of MCMI- III whether these signs are transient, developmental or ingrained.Methods279 patients with alopecia universalis were ...
K.K. Pirkalani, Z. Talaee Rad
core   +1 more source

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