Results 171 to 180 of about 1,649,512 (376)

Interferon Alfa in the treatment of B-cell chronic lymphocytic leukemia [PDF]

open access: yes, 1994
Adolf, D.   +10 more
core   +1 more source

Alpha Interferon and Not Gamma Interferon Inhibits Salmonid Alphavirus Subtype 3 Replication In Vitro

open access: yesJournal of Virology, 2010
Cheng Xu   +5 more
semanticscholar   +1 more source

In Situ Engineering of Tumor Cells as Self‐Sustaining cDC1 Programming Factories for Effective Cancer Immunotherapy

open access: yesAdvanced Science, EarlyView.
The fused EV platform AS16‐EL@MPLA/p‐FX reprograms tumor cells into cytokine‐producing factories that continuously secrete XCL1 and FLT3L to recruit and differentiate cDC1s, while embedded MPLA activates them through TLR4 signaling and AS16 prevents their exhaustion, thereby enhancing antitumor immunity and establishing durable immune memory against ...
Shuiling Jin   +16 more
wiley   +1 more source

Anterior ischemic optic neuropathy secondary to interferon alpha

open access: yesZdravniški Vestnik, 2005
Background: Anterior ischemic optic neuropathy rarely occurs with interferon alpha therapy. The mechanism by which interferon induces anterior ischemic optic neuropathy is unknown.Methods: A 62-year-old man presented with complaints of sudden severe loss
Stanka Godina-Kariž   +2 more
doaj  

Exosomes Exploit the Virus Entry Machinery and Pathway To Transmit Alpha Interferon-Induced Antiviral Activity. [PDF]

open access: yesJ Virol, 2018
Yao Z   +11 more
europepmc   +1 more source

Relationships between Quantitative Electroencephalographic Alterations and the Severity of Hepatitis C Based on Liver Biopsy in Interferon-.ALPHA. Treated Patients

open access: bronze, 2009
Satoshi Kamei   +13 more
openalex   +2 more sources

Ligand-Stimulated Downregulation of the Alpha Interferon Receptor: Role of Protein Kinase D2

open access: yesMolecular and Cellular Biology, 2010
Hui Zheng   +4 more
semanticscholar   +1 more source

Tumor‐Derived Interleukin 35 Promotes Fibrosis in the Tumor Microenvironment of Pancreatic Cancer by Activating Pancreatic Stellate Cells

open access: yesAdvanced Science, EarlyView.
This study demonstrates that tumor‐derived IL‐35 promotes pancreatic cancer fibrosis by indirectly activating pancreatic stellate cells through IGFBP2/IGF‐1R/PI3K‐Akt and Tsp‐1/TGF‐β pathways. IL‐35 blockade reduces stromal fibrosis, restores chemosensitivity, and synergizes with gemcitabine‐based regimens, highlighting IL‐35 as a promising therapeutic
Hui Li   +14 more
wiley   +1 more source

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