Results 231 to 240 of about 890,618 (298)
Prioritized Aczel-Alsina aggregation operators under p, q-quasirung orthopair fuzzy environment for sustainable supplier selection in new energy vehicle industry. [PDF]
Ali J, Al-Kenani AN, Syam MI.
europepmc +1 more source
Genomic basis underlying alternative transcripts-mediated drought tolerance in maize
Shulin Hao +9 more
openalex +1 more source
CNG: An alternative fuel for public transport [PDF]
Santosh A. Jalihal, T. Srinivasa Reddy
openalex
We show that the majority of the 18 analyzed recurrent cancer‐associated ERBB4 mutations are transforming. The most potent mutations are activating, co‐operate with other ERBB receptors, and are sensitive to pan‐ERBB inhibitors. Activating ERBB4 mutations also promote therapy resistance in EGFR‐mutant lung cancer.
Veera K. Ojala +15 more
wiley +1 more source
Selection of Base Materials for Repair Welding Using BWM-TOPSIS and BWM-RADAR Approaches. [PDF]
Arsić D +4 more
europepmc +1 more source
Peroxidasin enables melanoma immune escape by inhibiting natural killer cell cytotoxicity
Peroxidasin (PXDN) is secreted by melanoma cells and binds the NK cell receptor NKG2D, thereby suppressing NK cell activation and cytotoxicity. PXDN depletion restores NKG2D signaling and enables effective NK cell–mediated melanoma killing. These findings identify PXDN as a previously unrecognized immune evasion factor and a potential target to improve
Hsu‐Min Sung +17 more
wiley +1 more source
Rethinking Matrigel: The Complex Journey to Matrix Alternatives in Organoid Culture. [PDF]
Wolff L, Hendrix S.
europepmc +1 more source
This study shows that copy number variations (CNVs) can be reliably detected in formalin‐fixed paraffin‐embedded (FFPE) solid cancer samples using ultra‐low‐pass whole‐genome sequencing, provided that key (pre)‐analytical parameters are optimized.
Hanne Goris +10 more
wiley +1 more source
Single circulating tumor cells (sCTCs) from high‐grade serous ovarian cancer patients were enriched, imaged, and genomically profiled using WGA and NGS at different time points during treatment. sCTCs revealed enrichment of alterations in Chromosomes 2, 7, and 12 as well as persistent or emerging oncogenic CNAs, supporting sCTC identity.
Carolin Salmon +9 more
wiley +1 more source

