The target of rapamycin (TOR) complex is a central plant growth regulator. Here, the protein–protein interactions underlying its various functions are mapped by pupylation‐based proximity‐labeling in Arabidopsis, using TOR complex subunits LST8‐1 and RAPTOR1, alongside TOR inhibitor FK506‐binding protein, as baits.
Shuai Zheng+9 more
wiley +1 more source
Regulatory Mechanisms of Exogenous Acyl-Homoserine Lactones in the Aerobic Ammonia Oxidation Process Under Stress Conditions. [PDF]
Qiu C+6 more
europepmc +1 more source
ON THE DETECTION AND PRESERVATION OF CRYSTALLINE DEPOSITS OF URIC ACID, URATE OF AMMONIA, PHOSPHATE OF LIME, TRIPLE PHOSPHATE, OXALATE OF LIME, AND OTHER SALTS. [PDF]
Arthur Hassall
openalex +1 more source
Drug‐induced renal damage impairs uric acid excretion, causing its accumulation in the serum, which subsequently infiltrates the cochlea. Xanthine oxidase and the ABCG2 transporter, critical in uric acid metabolism and excretion, undergo pathological changes in renal tubular and cochlear stria vascularis cells.
Suhan Guo+11 more
wiley +1 more source
Red Mud Potentially Alleviates Ammonia Nitrogen Inhibition in Swine Manure Anaerobic Digestion by Enhancing Phage-Mediated Ammonia Assimilation. [PDF]
Peng Y+13 more
europepmc +1 more source
RECOVERY AFTER HANGING, BY MEANS OF BLEEDING AND AMMONIA LINIMENT. [PDF]
John J. Johnston
openalex +1 more source
SOLUTIONS OF METALS IN NON-METALLIC SOLVENTS; I. GENERAL PROPERTIES OF SOLUTIONS OF METALS IN LIQUID AMMONIA. [PDF]
Charles A. Kraus
openalex +1 more source
Development of MDM2‐Targeting PROTAC for Advancing Bone Regeneration
This work presents a creative pipeline for developing MDM2‐targeting PROTACs (MDM2‐PROTACs) for application in bone regeneration. The developed PROTACs (CL144 and CL174) are validated for their degradation efficiency and osteogenic effects in human BMSCs.
Sol Jeong+17 more
wiley +1 more source
Pressure Swing Absorption and Desorption of Ammonia in Calcium Halide Solid Mixtures
Manabu Tokushige+3 more
doaj +1 more source
The study reveals that glutaminolysis in macrophages is inhibited under type 2 diabetes mellitus (T2DM) conditions, which impedes fracture healing by reducing bone morphogenetic protein 2 (BMP2) production through increased cytosine methylation on the promoter.
Jing Wang+12 more
wiley +1 more source