Results 231 to 240 of about 54,677 (265)
Dual adaptive strategies in <i>Candida glabrata</i> under tunicamycin stress: petite mutations and chromosome C aneuploidy drive transient drug resistance. [PDF]
Front MicrobiolDong Y +5 more
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Physiological Response of Human Cells to Aneuploidy - Transcriptome Studies and Cellular Assays
, 2012 Milena Dürrbaum
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Evolutionary rescue by aneuploidy in tumors exposed to anticancer drugs. [PDF]
GeneticsStana R +3 more
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Hidden in Plain Sight: A Rare 7q Deletion Masquerading as a Common Aneuploidy on Prenatal Ultrasound. [PDF]
CureusAbang Abdullah ZH +4 more
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Live Motile Sperm Sorting Device Improves Embryo Aneuploidy: A Retrospective Cohort Study
K. E. Gotsiridze +3 more
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Journal of Intellectual Disability Research, 1980
On one hundred consecutive clinically referred patients, a total of 3,175 cells was fully analysed. The parameters of culture and analysis were the same for all patients, and a breakdown of cells showed 258 cells (8.1 per cent) with hypomodal counts and sixty-two cells (1.9 per cent) with hypermodal counts.
A, Smith, G, Elliott
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On one hundred consecutive clinically referred patients, a total of 3,175 cells was fully analysed. The parameters of culture and analysis were the same for all patients, and a breakdown of cells showed 258 cells (8.1 per cent) with hypomodal counts and sixty-two cells (1.9 per cent) with hypermodal counts.
A, Smith, G, Elliott
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Current Opinion in Obstetrics and Gynaecology, 1999
Over the past 15 years, biochemical screening for chromosomal abnormalities, particularly Down's syndrome, has advanced from being extremely naive, to now somewhat more sophisticated. Sensitivities have gone from 20% to 60-70%. Considerable work is still required to not only increase the sensitivity, but also the specificity to keep health care costs ...
M I, Evans, J E, O'Brien, A, Johnson
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Over the past 15 years, biochemical screening for chromosomal abnormalities, particularly Down's syndrome, has advanced from being extremely naive, to now somewhat more sophisticated. Sensitivities have gone from 20% to 60-70%. Considerable work is still required to not only increase the sensitivity, but also the specificity to keep health care costs ...
M I, Evans, J E, O'Brien, A, Johnson
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Journal of Cellular Biochemistry, 2007
AbstractThe cell's euploid status is influenced by, amongst other mechanisms, an intact spindle assembly checkpoint (SAC), an accurate centrosome cycle, and proper cytokinesis. Studies in mammalian cells suggest that dysregulated SAC function, centrosome cycle, and cytokinesis can all contribute significantly to aneuploidy.
Ya-Hui, Chi, Kuan-Teh, Jeang
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AbstractThe cell's euploid status is influenced by, amongst other mechanisms, an intact spindle assembly checkpoint (SAC), an accurate centrosome cycle, and proper cytokinesis. Studies in mammalian cells suggest that dysregulated SAC function, centrosome cycle, and cytokinesis can all contribute significantly to aneuploidy.
Ya-Hui, Chi, Kuan-Teh, Jeang
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Reviews in Medical Virology, 2019
SummarySeven oncogenic viruses are known for tumorigenesis and contribute to 12% of all human cancers. The oncogenic factors, the target tissue, and pathology of cancer vary among these viruses with several mechanisms proposed for the initiation and development of cancer. Aneuploidy in cells is associated with anomalies in chromosome number that can be
Fateme Taheri +2 more
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SummarySeven oncogenic viruses are known for tumorigenesis and contribute to 12% of all human cancers. The oncogenic factors, the target tissue, and pathology of cancer vary among these viruses with several mechanisms proposed for the initiation and development of cancer. Aneuploidy in cells is associated with anomalies in chromosome number that can be
Fateme Taheri +2 more
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Nature, 2004
In contrast to normal cells, aneuploidy--alterations in the number of chromosomes--is consistently observed in virtually all cancers. A growing body of evidence suggests that aneuploidy is often caused by a particular type of genetic instability, called chromosomal instability, which may reflect defects in mitotic segregation in cancer cells.
Harith, Rajagopalan, Christoph, Lengauer
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In contrast to normal cells, aneuploidy--alterations in the number of chromosomes--is consistently observed in virtually all cancers. A growing body of evidence suggests that aneuploidy is often caused by a particular type of genetic instability, called chromosomal instability, which may reflect defects in mitotic segregation in cancer cells.
Harith, Rajagopalan, Christoph, Lengauer
openaire +2 more sources