Results 21 to 30 of about 382,630 (241)

Management of unstable angina [PDF]

open access: yesDrug and Therapeutics Bulletin, 1998
Unstable angina (defined as attacks that are increasingly frequent and/or prolonged, that occur at rest, or are brought on by trivial provocation) is one of the commonest reasons for emergency admission to hospitals in the UK. Even with appropriate treatment, the risk of myocardial infarction (MI) or death may be as high as 10% in the first 6 weeks ...
openaire   +2 more sources

Progression of Coronary Artery Disease (CAD) from Stable Angina (SA) Towards Myocardial Infarction (MI): Role of Oxidative Stress [PDF]

open access: yesJournal of Clinical and Diagnostic Research, 2014
Introduction: There is now a consensus that atherosclerosis represents a state of heightened oxidative stress which is characterized by lipid and protein oxidation in the vascular wall.
Neha Uppal, Vibha Uppal, Pankaj Uppal
doaj   +1 more source

Histopathological analysis of the coronary atheroma extracted during coronary artery bypass graft surgery

open access: yesBangabandhu Sheikh Mujib Medical University Journal, 2018
This study aims to evaluate the histopathological analysis as well as the effect of coronary endarterectomy with severe calcified coronary artery disease.
Redoy Ranjan   +5 more
doaj   +1 more source

Pathogen burden, inflammation, proliferation and apoptosis in human in-stent restenosis - Tissue characteristics compared to primary atherosclerosis [PDF]

open access: yes, 2004
Pathogenic events leading to in-stent restenosis (ISR) are still incompletely understood. Among others, inflammation, immune reactions, deregulated cell death and growth have been suggested. Therefore, atherectomy probes from 21 patients with symptomatic
Wernert, N.   +7 more
core   +1 more source

SELL and GUCY1A1 Gene Polymorphisms in Patients with Unstable Angina

open access: yesBiomedicines, 2022
Acute ischaemia is mostly caused by the rupture of an unstable atherosclerotic plaque in a coronary artery, resulting in platelet accumulation and thrombus formation, which closes the lumen of the coronary vessel.
Damian Malinowski   +4 more
doaj   +1 more source

Periodontitis and atherosclerotic cardiovascular disease: A critical appraisal

open access: yesPeriodontology 2000, EarlyView., 2023
Abstract In spite of intensive research efforts driving spectacular advances in terms of prevention and treatments, cardiovascular diseases (CVDs) remain a leading health burden, accounting for 32% of all deaths (World Health Organization. “Cardiovascular Diseases (CVDs).” WHO, February 1, 2017, https://www.who.int/news‐room/fact‐sheets/detail ...
Maria Clotilde Carra   +3 more
wiley   +1 more source

Coexistence of Unstable Angina Pectoris and Wolff-Parkinson-White Syndrome Developed After Consumption of Energy Drink

open access: yes, 2018
Interest in energy drinks and their consumption are increasing in recent years. The underlying motivation for the users is mostly enjoyment or enhancement of performance and attentiveness.
Hüseyin Ede   +4 more
core   +1 more source

PATHOPHYSIOLOGY, INVESTIGATIONS, AND MANAGEMENT OF UNSTABLE ANGINA: A REVIEW [PDF]

open access: yes, 2022
Unstable angina pectoris is a phenomenon that disrupts the early recovery phase of acute myocardial infection, the emergence of new ischemic symptoms, the development of more severe pain, usually at rest, or the formation of intermittent ischemic events.
ADEL GAMIL, MOHAMED   +5 more
core   +1 more source

Unstable Angina: A Classification [PDF]

open access: yesCirculation, 1989
The establishment of a prognosis and the approach to the treatment of many diseases is aided greatly by a logical classification. For example, classification of a wide variety of neoplasms by anatomic extent, microscopic appearance, and the presence of special markers now forms the basis for selecting appropriate therapy.
openaire   +2 more sources

Unstable angina pectoris

open access: yesClinical Cardiology, 1990
AbstractThe pathophysiology of unstable angina has been better elucidated in the past five years and has led to more rational therapy. Coronary arteries in patients with unstable angina have atherosclerotic plaques which are often complex and are the site of platelet activation and fibrin deposition.
W A, Wallace, J F, Richeson, P N, Yu
openaire   +2 more sources

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