Results 201 to 210 of about 1,463,025 (368)

Systemic autoimmune rheumatic disease risk is associated with long‐term exposure to fine particulate matter

open access: yesArthritis &Rheumatology, Accepted Article.
Objective Fine particulate matter (PM2.5) is a possible trigger of systemic autoimmune rheumatic diseases (SARDs). We investigated SARDs risk related to long‐term exposure to PM2.5 and its components (ammonium, black carbon, mineral dust, sea salt, nitrate, sulfate, organic matter), the composition of which may affect toxicity.
Mareva Geslin   +6 more
wiley   +1 more source

Ants

open access: yes, 2003
Biblioteca de Educación del Ministerio de Educación, Cultura y Deporte; Calle San Agustín, 5 - 3 planta; 28014 Madrid; Tel.
openaire   +1 more source

The Role of the Brain's Pragmatic Language Network in Reading Comprehension in Autistic Children

open access: yesAutism Research, EarlyView.
ABSTRACT One of the earliest and commonly reported symptoms of autism spectrum disorder (ASD) is a delay in language development. Such delay may sometimes accompany deficits which can have a long‐term impact on reading comprehension. It is frequently reported that autistic children exhibit significant difficulties in pragmatics, which is the ...
Elizabeth Valles‐Capetillo   +2 more
wiley   +1 more source

Sodium Manganese Hexacyanoferrate: Characterization as Sodium‐Ion Battery Cathode Material, Full Cell Cycling with Hard Carbon and Post‐Mortem Analyses

open access: yesBatteries &Supercaps, EarlyView.
Sodium manganese hexacyanoferrate (NaMnHCF) is a promising cathode for sodium‐ion batteries due to its LFP‐like voltage profile and cost‐effective composition. This study examines its cycling performance with hard carbon anodes, highlighting Na+ inventory loss as a key degradation factor.
Sebastian Büchele   +15 more
wiley   +1 more source

Reduced Adult Neurogenesis in Humans Results From a Tradeoff Rather Than Direct Negative Selection

open access: yesBioEssays, EarlyView.
Embryonic radial glia (RG) generate neurons, glial cells, and later adult neural stem cells, which sustain adult neurogenesis (top, left to right). Human‐specific genetic modifications (blue arrow), selected to boost early RG neurogenic activity, may lead to premature RG exhaustion and reduced adult neurogenesis in the human brain (bottom).
David Morizet, Laure Bally‐Cuif
wiley   +1 more source

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