Infectious bacteria as biological warfare agents: mechanisms, epidemiological threats, and defense strategies. [PDF]
Shah K, Khan MI, Guo Y, Adnan M, Wu H.
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Laboratory Diagnostics of Botulism Cases in Livestock in Poland in 2022-2024. [PDF]
Jarosz A, SapaĆa M, Grenda T.
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Mono- and multidomain defense toxins of the RelE/ParE superfamily. [PDF]
Gerdes K.
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Genomic profiling of <i>Morganella morganii</i>: insights into antibiotic resistance genes, pathogenicity islands, and toxin-antitoxin systems. [PDF]
Senbadejo TY, Bourne CR, Isawumi A.
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Bistability in type I toxin-antitoxin systems may lead to stress-induced persister formation. [PDF]
Markovic S +3 more
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Ser/Thr phosphorylation of <i>Mycobacterium tuberculosis</i> type II RelK toxin by PknK destabilizes TA interaction and interferes with toxin neutralization. [PDF]
Sarah SR +6 more
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Single Tri-Epitopic Antibodies (TeAbs) to Botulinum Neurotoxin Serotypes B, E, and F Recapitulate the Full Potency of a Combination of Three Monoclonal Antibodies in Toxin Neutralization. [PDF]
Lou J +6 more
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Recent genomic analyses revealed a surprisingly large number of toxin-antitoxin loci in free-living prokaryotes. The antitoxins are proteins or antisense RNAs that counteract the toxins. Two antisense RNA-regulated toxin-antitoxin gene families, hok/sok and ldr, are unrelated sequence-wise but have strikingly similar properties at the level of gene and
Kenn Gerdes, Gerhart Wagner
exaly +4 more sources
This study provides evidence that an antisense RNA antitoxin is involved in persister cell formation by inhibiting transcription of a toxin and by inhibiting global translation.
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