Results 251 to 260 of about 10,664,685 (382)
Conservative drug treatment in patients with moderately severe chronic occlusive peripheral arterial disease. Scandinavian Study Group. [PDF]
Folke Lindgärde+6 more
openalex +1 more source
Cardiovascular disease (CVD) and cancer are leading causes of death worldwide, with overlapping risk factors and pathophysiological mechanisms. This review explores shared pathways, including metabolic dysregulation, chronic inflammation, and gut microbiome alterations, highlighting dual‐benefit strategies such as lifestyle modifications and repurposed
Shihan Xiang+6 more
wiley +1 more source
Extracorporeal shock wave therapy for peripheral arterial disease-related intermittent claudication: a systematic review and meta-analysis. [PDF]
Tang P+8 more
europepmc +1 more source
THE PATHOLOGY OF ARTERIAL DISEASE
A.G. Maciver, P.J. Gallagher
openaire +3 more sources
Occlusive thromboaortopathy (Takayasu's disease): cervical arterial stenoses, retinal arterial pressure, retinal microaneurysms and prognosis. [PDF]
K. Ishikawa, M Uyama, K Asayama
openalex +1 more source
This study reveals that the glycolytic enzyme HK2 mediates hypoxia‐induced astrocytic exosome biogenesis by directly phosphorylating nSMase1. These astrocytic exosomes impair brain endothelial tight junctions. Astrocyte‐specific knockdown of HK2 reduces exosome release and alleviates brain injury after MCAO.
Chen Chen+12 more
wiley +1 more source
Temporal changes in symptomatic intracranial arterial disease: a longitudinal high-resolution vessel wall imaging study. [PDF]
Kang DW+9 more
europepmc +1 more source
Multiple progressive intracranial arterial occlusions (`moyamoya' disease)
D J O'Sullivan+3 more
openalex +1 more source
Fluid Mechanics, Arterial Disease, and Gene Expression.
J. Tarbell+3 more
semanticscholar +1 more source
This study highlights GRK2 is a central mediator in OSS‐induced endothelial dysfunction. OSS activates GPCRs in endothelial cells, leading to GRK2 phosphorylation and the activation of AP‐1. AP‐1 induces inflammation, while also promoting NR4A1 expression and anchoring LKB1 in the nucleus, which suppresses AMPK activity. This cascade causes endothelial
Li‐Da Wu+18 more
wiley +1 more source