Results 231 to 240 of about 293,921 (283)

Articular cartilage transplantation

Human Pathology, 1977
This report describes the biopsy findings in four of 30 patients treated with cadaver osteochondral shell allografts for osteoarthritis in the knee. This study demonstrates that graft cartilage cells can survive in excess of 25 months, and that host bone can completely replace graft bone by creeping substitution.
K P, Pritzker, A E, Gross, F, Langer, S C, Luk, J B, Houpt   +4 more
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Permeability of Articular Cartilage

Nature, 1968
IN 1962, McCutcheon1 first drew attention to a decrease in the permeability of articular cartilage with increasing depth from the surface. This observation has been confirmed2 and the permeability shown to be inversely related to the fixed charge density of the cartilage matrix.
A, Maroudas, P, Bullough
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The Microhardness of Articular Cartilage

Clinical Orthopaedics and Related Research, 1975
The standard metallurgical technique of microhardness testing was useful for investigations on the physical properties of articular cartilage. The problem of visco-elasticity of the cartilage was overcome by using a brittle lacquer coating as a memory device. The surface layer was the hardest plane when the superficial layer was intact.
H U, Cameron, R M, Pillar, I, Macnab
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Fracture of Articular Cartilage

Journal of Biomechanical Engineering, 1996
Crack formation and propagation is a significant element of the degeneration process in articular cartilage. In order to understand this process, and separate the relative importance of structural overload and material failure, methods for measuring the fracture toughness of cartilage are needed.
M V, Chin-Purcell, J L, Lewis
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CALCIFICATION IN ARTICULAR CARTILAGE

The Journal of Bone and Joint Surgery. British volume, 1960
A case of calcification of articular cartilage in association with a parathyroid tumour is described. Previously reported cases of articular calcification are briefly discussed, and it is recommended that patients with articular calcification of undetermined cause should be investigated for hyperparathyroidism.
G E, HOSKING, G, CLENNAR
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Regeneration of Articular Cartilage

2005
Loss of articular cartilage from the ends of bones forming diarthrodial joints can be the source of profound pain and disability, and eventually lead to complete degeneration of the joint, necessitating total joint replacement. Until a few years ago, there seemed little hope of treating such defects.
B, Kinner, R M, Capito, M, Spector
openaire   +2 more sources

Integrins in Articular Cartilage

2008
In the following sections, we primarily review our own work on the expression, tissue distribution and function of integrins in selected in vitro models of articular chondrocytes and limb bud mesenchymal cells. The expression pattern of α1-, α3-, αv- and α5β1-integrins and their specific ligand binding were investigated in monolayer cultures of ...
Shakibaei, M, Csaki, C, Mobasheri, A
openaire   +3 more sources

Fatigue of Articular Cartilage

Nature, 1973
IT has been suggested1 that fibrillation, the earliest change in osteoarthritic cartilage visible to the naked eye, may be the result of fatigue failure. Abnormally high stresses in the superficial layer of cartilage could be produced by unusually high applied loads, incongruity of the joint surface, or softening of the cartilage by mucopolysaccharide ...
B O, Weightman, M A, Freeman, S A, Swanson   +2 more
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Ultrastructure of articular cartilage

Zeitschrift f�r Zellforschung und Mikroskopische Anatomie, 1959
The present paper describes an electron microscopical investigation into the ultrastructure of patellary and femorocondylar articular cartilage from adult guinea pigs, mice and young rats. The following observations were made: 1. The intercellular matrix contains two varieties of collagen fibrils, one being 85 A in diameter and not striated, the ...
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Articular Cartilage Biology

Journal of the American Academy of Orthopaedic Surgeons, 2003
Articular cartilage is a complex tissue maintained by chondrocytes, which undergo metabolic changes as a result of aging, disease, and injury. These changes may hinder tissue maintenance and repair, resulting in accelerated loss of articular surface and leading to end-stage arthritis.
Michael, Ulrich-Vinther, Michael D, Maloney, Edward M, Schwarz, Randy, Rosier, Regis J, O'Keefe   +4 more
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