Results 271 to 280 of about 202,771 (390)
Metabolite profiling to evaluate metabolic changes in drought-tolerant transformant (Agb0103) under salt stress. [PDF]
Lee GM +9 more
europepmc +1 more source
Active-site-directed inhibition of asparagine N-glycosyltransferases with epoxy-peptide derivatives [PDF]
Ernst Bause
openalex +1 more source
The potential for biased signalling in the P2Y receptor family of GPCRs
The purinergic receptor family is primarily activated by nucleotides, and contains members of both the G protein coupled‐receptor (GPCR) superfamily (P1 and P2Y) and ligand‐gated ion channels (P2X). The P2Y receptors are widely expressed in the human body, and given the ubiquitous nature of nucleotides, purinergic signalling is involved with a plethora
Claudia M. Sisk +2 more
wiley +1 more source
The Landscape of Cancer Metabolism as a Therapeutic Target. [PDF]
Ohshima K.
europepmc +1 more source
Reprimo (RPRM): A Tumor Suppressor That Induces Extrinsic Apoptosis via YAP Signaling
Reprimo (encoded by RPRM) is a p53 target gene that acts as a tumor suppressor and is frequently silenced by promoter hypermethylation in various cancers. Although previously known to function intracellularly as a tumor suppressor, recent studies revealed that secreted Reprimo also induces apoptosis in neighboring cells through a novel p53‐Reprimo ...
Masahiro Takikawa, Rieko Ohki
wiley +1 more source
Deciphering the metabolic patterns of cashew apple ripening process: A comprehensive non-targeted metabolomics analysis. [PDF]
Huang H +10 more
europepmc +1 more source
Purification of hamster melanoma tyrosinases and structural studies of their asparagine-linked sugar chains [PDF]
Takashi Ohkura +3 more
openalex +1 more source
GLS1‐mediated glutaminolysis supports GSH synthesis in cancer cells. GLS1 KO increases ROS, downregulates GPX4, and upregulates GPX1, making cells more sensitive to ferroptosis. Combining GLS1 or GPX1 inhibitors with a GPX4 inhibitor synergistically suppresses cancer growth.
Changsen Bai +13 more
wiley +1 more source

